using a questionnaire pre-established by Google forms whose link was shared on social networks. Medical students who validated 5 years of study or GPs in Senegal and agreeing to participate were included. Results: Sixty-nine respondents (27.13 AE 1.7 years old, 60.9% men) were selected for the analysis. They were in doctoral year in 58%, in 7th year of medicine in 30.4%, GPs in 5.8%. Among the main causes of the CKD, diabetes and hypertension were cited together by 52.5% of respondents.Conclusions: It would be important to strengthen the knowledge and practices of end-of-cycle students and general practitioners by promoting continuing medical education on the CKD and the evaluation of professional practices.

Conclusions: Lithium nephrotoxicity spans a spectrum, from concentration defects to nephrogenic diabetes insipidus, chronic interstitial nephritis and rare glomerular involvement. Duration of treatment and cumulative dose are the determinants of renal toxicity. Careful monitoring and early withdrawal are the key to limiting damage, as a creatinine more than 2.5mg/dl at biopsy is a risk for progression to ESRD. In those with early signs of toxicity, but in whom Lithium would be necessary, a concurrent use of Amiloride could limit the damage.This case was presented to highlight the diagnosis of a form of Lithium toxicity in the form of Chronic interstitial nephritis and polyuria that was incidentally picked up on evaluation for a different complaint. The fact that he had polyuria appropriate to his intake, which subsequently corrected when intake was restricted, suggested a psychogenic polydipsia. The patient would be benefitted by being cautioned not to be exposed to lithium in future.