We consider a Keller–Segel model that describes the cellular chemotactic movement away from repulsive chemical subject to logarithmic sensitivity function over a confined region in
${{\mathbb{R}}^n},\,n \le 2$
. This sensitivity function describes the empirically tested Weber–Fecher’s law of living organism’s perception of a physical stimulus. We prove that, regardless of chemotaxis strength and initial data, this repulsive system is globally well-posed and the constant solution is the global and exponential in time attractor. Our results confirm the ‘folklore’ that chemorepulsion inhibits the formation of non-trivial steady states within the logarithmic chemotaxis model, hence preventing cellular aggregation therein.
<p style='text-indent:20px;'>One of the most impressive findings in chemotaxis is the aggregation that randomly distributed bacteria, when starved, release a diffusive chemical to attract and group with others to form one or several stable aggregates in a long time. This paper considers pattern formation within the minimal Keller–Segel chemotaxis model with a focus on the stability and dynamics of its multi-spike steady states. We first show that any steady-state must be a periodic replication of the spatially monotone one and they present multi-spikes when the chemotaxis rate is large; moreover, we prove that all the multi-spikes are unstable through their refined asymptotic profiles, and then find a fully-fledged hierarchy of free entropy energy of these aggregates. Our results also complement the literature by finding that when the chemotaxis is strong, the single boundary spike has the least energy hence is the most stable, the steady-state with more spikes has larger free energy, while the constant has the largest free energy and is always unstable. These results provide new insights into the model's intricate global dynamics, and they are illustrated and complemented by numerical studies which also demonstrate the metastability and phase transition behavior in chemotactic movement.</p>
This paper investigates the Keller-Segel model with quadratic cellular diffusion over a disk in R 2 with a focus on the formation of its nontrivial patterns. We obtain explicit formulas of radially symmetric stationary solutions and such configurations give rise to the ring patterns and concentric airy patterns. These explicit formulas empower us to study the global bifurcation and asymptotic behaviors of these solutions, within which the cell population density has δ-type spiky structures when the chemotaxis rate is large. The explicit formulas are also used to study the uniqueness and quantitative properties of nontrivial stationary radial patterns ruled by several threshold phenomena determined by the chemotaxis rate. We find that all nonconstant radial stationary solutions must have the cellular density compactly supported unless for a discrete sequence of bifurcation values at which there exist strictly positive small-amplitude solutions. The hierarchy of free energy shows that in the radial class the inner ring solution has the least energy while the constant solution has the largest energy, and all these theoretical results are illustrated through bifurcation diagrams. A natural extension of our results to R 2 yields the existence, uniqueness and closedform solution of the problem in this whole space. Our results are complemented by numerical simulations that demonstrate the existence of non-radial stationary solutions in the disk.
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