BackgroundThe incidence of skin cancer has increased worldwide, particularly melanoma rates, which had a mean development of 2.6 % a year in the last 10 years. The agreement on the relation between long-term or chronic exposure to the sun and the emergence of these neoplasias has made several workers who perform activities exposed to solar radiation to form a risk group for the development of skin cancer, community health agents included.OBJECTIVESTo analyze the prevalence of sunscreen-use-related factors to skin cancer in a labor risk group.METHODOLOGYCross-sectional study with community health agents selected through simple random sampling. After collecting data using semi-structured interviews, a descriptive analysis was performed for the qualitative variables, bivariate analysis was employed for checking the association between sunscreen use and sociodemographic, occupational and knowledge about skin variables, and multivariate analysis was conducted to check independent variables associated to sunscreen use. A 5% significance level was used.ResultsOf 261 health gents selected, 243 were able to participate in the study. The prevalence rate of sunscreen use was 34.2% (95% CI: 28.2-40.2). Factors associated with sunscreen use were female sex, advanced age, use of sunscreen in situations when the skin got burnt, knowledge of the negative effects of the sun on the skin and skin cancer history.ConclusionsThe prevalence found reveals that there is a need for implementing educational strategies in health services regarding photoprotection.

Phenol-croton oil peel enhances type-1 and type-3 collagen amounts by stimulating SIRT-6 and SIRT-7Dear Editor, Adults lose 1% of collagen per year because of aging. 1This process impacts negatively more on type-3 (COL3) than type-1 (COL1) collagen and is aggravated by metalloproteinases and photoaging-decreased neocollagenesis. 2Sirtuins (SIRT), a histone-deacetylase family, cause longevity of fibroblasts and regulate collagen synthesis. 3 SIRT protect skin against UV-dependent damage by repairing DNA, controlling inflammation, and modulating antioxidant defense. 4 SIRT-1,

Phenol-croton oil peel generates skin neocollagenesis. Combination of 35% phenol and 0.4% to 1.6% croton oil creates formulas that vary in intensity and penetration capacity, being used for different degrees of skin aging and application areas, with low demarcation and systemic risk. 1,2 An open, longitudinal, non-placebo-controlled study was conducted with 30 women (52 to 87 years old; Fitzpatrick I to IV) with moderate-to-severe perioral aging (Scales of Glogau≥III, Baker≥II, and Cohen≥moderate), excluding those with systemic diseases and 12-month-previously perioral esthetic procedure. SPF60 was used for 30 days pre-peeling and acyclovir 400mg, Q4H, for 7 days postpeeling. Forty-mg codeine was administered 1 hour before and maintained BID for 2 days post-procedure.Before (D0) and 90±2days after (D90±2) the procedure, photographs and punch-n 0 4 biopsies were performed (D0, 0.5cm, laterally to the left filtrum midpoint; D90±2, 0.5cm laterally to the D0's).

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