Approximately 60% of Australia’s beef cattle are located in the vast rangelands of northern Australia. Despite the often low stocking densities and extensive management practices of the observed herd, animal prevalence of BVDV infection and typical rates of transmission are similar to those observed in intensively managed herds in southern Australia and elsewhere in the world. A recent large three- to four-year study of factors affecting the reproductive performance of breeding herds in this region found that where there was evidence of widespread and/or recent BVDV infection, the percentage of lactating cows that became pregnant within four months of calving was reduced by 23%, and calf wastage was increased by 9%. BVDV is now considered the second most important endemic disease affecting beef cattle in northern Australia, costing the industry an estimated AUD 50.9 million annually. Although an effective killed vaccine was released in Australia in 2003, the adoption of routine whole herd vaccination by commercial beef farmers has been slow. However, routine testing to identify persistently infected replacement breeding bulls and heifers has been more widely adopted.

Epitheliogenesis imperfecta (EI) has been diagnosed in calves on a north Queensland beef cattle research station. There are five Bos indicus cross, inter se mated breed groups on the station: 1/2 Brahman, 3/4 Brahman, 1/2 Sahiwal, 3/4 Sahiwal and High Grade Sahiwal. The disease was confined to the three Sahiwal breed groups and caused calf losses over seven years from 1978 of 0.2%, 3.4% and 1.0% in the 1/2, 3/4 and High Grade groups respectively. The Sahiwal breed was identified as the origin of the gene responsible for the disease. The source of the gene within the Sahiwal breed was investigated, and several ancestors were identified as almost certainly being heterozygotes. Analyses of relationships of affected and non-affected calves to the 10 Sahiwals that were originally imported into Australia identified the one that was most likely the source of the El gene.