Four individuals mistook Podostroma cornu-damae for Cordyceps sobolifera and reported diarrhoea and vomiting after consuming the toxic mushroom. All symptoms resolved within four days. However, leukocytopenia, thrombocytopenia, depilation, and desquamation appeared later, and one patient was diagnosed with sepsis. All patients received granulocyte colony-stimulating factor and blood transfusions in an isolated aseptic room, and they recovered without complications. Podostroma cornu-damae is a poisonous mushroom containing trichothecene. This mushroom is not well known and is easy to be mistaken for the more common Cordyceps sobolifera sold as a healthy food in East Asia. Podostroma cornu-damae intoxication has characteristic early and delayed symptoms. Early symptoms are mainly gastrointestinal, while delayed symptoms are caused by inhibition of cellular division causing pancytopenia and can lead to death. A case series of Podostroma cornu-damae poisoning was presented, including one complicated case complicated with life-threatening sepsis. (Hong Kong j.emerg.med. 2016;23:55-59) 四個人誤以火焰茸為蟬花，服用毒蘑菇後出現腹瀉和嘔吐。所有症狀在四天之內消失。然而隨後出現白 細胞減少、血小板減少、脫毛和脫屑，其中一個病人被診斷為敗血症。所有患者在一個獨立的無菌室接 受粒細胞集落刺激因子和輸血後，全都能康復且無併發症。火焰茸是一種含有單端孢的有毒蘑菇。這種 蘑菇不是廣為人知，很容易被誤認為是蟬花，一種在東亞較為常見的健康食品。火焰茸中毒的特徵是具 有早期和延遲症狀。早期症狀主要是胃腸道的，而延遲症狀是由抑制細胞分裂引起的全血細胞減少引起 的，可導致死亡。我們報告一系列的火焰茸中毒病例，包括一例併發可危及生命的敗血症。

A 2-year-old mixed breed dog presented with a 1-year history of crust and erosion on the nasal planum. Because histopathological examination revealed ruptured intraepidermal pustules and superficial dermal inflammation, the dog was diagnosed with pemphigus foliaceus. Human intravenous immunoglobulin was administered in two consecutive doses of 0.5 g/kg/day due to poor therapeutic response to previous immunosuppressive therapy. From Day 3 after the first dose of human intravenous immunoglobulin, tachypnoea, pale mucous membrane, haemoglobinuria and haemoglobinemia were observed, thus confirming haemolytic anaemia. Other drug-induced haemolytic anaemias were excluded because no additional drugs had been administered before the haemolysis occurred. Immune-mediated haemolytic anaemia was also excluded because the direct antiglobulin test was negative. Two transfusions were performed, and haemolysis was not observed from Day 4 of haemolytic anaemia onset. In conclusion, human intravenous immunoglobulin-induced haemolytic anaemia should be considered in dogs that develop haemolysis following the administration of human intravenous immunoglobulin.