A factorial experiment with two factors was carried out at the Seed Technology Lab., College of Agriculture, University of Baghdad in 2018 in order to study the effect of seed priming with gibberellic acid (GA3) (0, 100, 300 and 500 mgL-1) on germination and seedling growth of deteriorated seed of two oat cultivars (Shifaa, and Shofan11). The complete randomized design was used with four replications. The results showed that cultivars, soaking in different concentrations of GA3 and their interactions had a significant effect on studied traits. Shofan11, GA3 (500 mg L-1) and their interaction gave the highest values of germination ratio (74.1, 85 and 85%), lengths of radical (7, 8.5 and 8.6 cm) and plumule (9.3, 10.8 and 10.9 cm), fresh (71.95, 74.74 and 74.76 mg) and dry (18.41, 23.14 and 25.07 mg) weights of seedling and seedling vigour index (1270.2, 1644 and 1655.5), respectively. It can be concluded that soaking of deteriorated seed with GA3 led to increasing germination ratio and improved seedling growth in oat.  

Breasts are one of the secondary sexual characteristics in females. They are the rout for of nutrition & growing of infant till 4-6 months of age. Breast diseases are one of the most common diseases in the females of any society. Multiple types of benign breast lesions like fibroadenomas, simple cyst, breast abscess, lymph nodes enlargement and different malignancies are common pathologies of the breast. Up to 30% of women suffer from benign breast disease at any time of their life and this compels them to take the treatment. The prospective clinical study was achieved for 283 women ( age was ranged between 20 60 years) had palpable breast lesion referred by their managing surgeons to the radiological department at Azadi Teaching Hospital, for a period of 8 months (from January 2018 to August of the same year). The study depended firstly in all women on clinical examination/self-breast examination. Results:- 283 patients were included in this study with age between 20 _ 60 ys, most of the patients were diagnosed as fibroadenoma which is most common around 54.9% of the total cases studied, Fibrocystic diseases (Duct ectasia, cysts, and galactocele ) 19.3 %.Followed by infection as mastitis 15.4%, and phyllodes tumor less than 1% approximately and finally carcinoma 9.5%.

Background: Myoadenylate deaminase deficiency is an autosomal recessive metabolic myopathy caused by mutations in the Adenosine monophosphate deaminase 1 gene. Adenosine monophosphate deaminase 1 gene deficiency is one of the most common causes of exercise-induced myopathy. In this study, non-synonymous single nucleotide polymorphism was analyzed for its functional and structural impact which is deleterious to Adenosine monophosphate deaminase 1 protein. Methods: The data on human Adenosine monophosphate deaminase 1gene was retrieved from the NCBI database on 9 JUNE 2021 and then analyzed using different bioinformatics prediction algorithms, namely: SIFT, PolyPhen-2, PROVEAN, SNAP2, PANTHER, SNPs and GO, PMut, and I-Mutant to detect the deleterious nsSNPs and its association with diseases. In addition, a Consurf web server was used to detect the functional SNPs in the conserved region. Chimera, Project Hope, and MutPred2 software were used to visualize and analyze the effect of nsSNPs on the functions and structure of the AMPD1 protein. Finally, both the STRING database and KEGG were used for the prediction of protein-protein interaction. Results: A total of 6178 SNPs were reported in the human AMPD1 gene. In this study 583 missense nsSNPs were selected for investigation and only 72 nsSNPs were shortlisted and computationally evaluated for their impact on AMPD1 protein. From all servers that were used collectively (K320I, R421W, R458C, R458H, R51C, R757L, R761H, and G246S) nsSNPs were predicted as deleterious, associated with disease, highly conserved, and decrease effective stability of AMPD1 protein. In addition, the AMPD1 protein was predicted to have strong interactions with ten proteins involved in various ranges of biological processes. Conclusion:The present study undertakes a systematic bioinformatics approach to identify functionally important nsSNPs in the human AMPD1 gene to understand how these mutations affect the protein function and structure and hence promote a myoadenylate deaminase deficiency.

The goal is to contribute towards well understanding the solid state reaction between TiC base ceramic and nano additives. A mixture of elemental powders of Ti and Graphite doped with a range of ceramic nano additives of each Al 2 O 3 and CuO was pressed and heated up to 1100 °C. Phase evolution was then investigated using X-ray diffraction (XRD) and Differential thermal analysis (DTA). TiC is identified to be the most dominant phase through the reaction between Ti and Graphite at 1100 °C. TiO 2 is the most detected oxide due to the reaction of Ti-C/ nano Al 2 O 3 and also for Ti-C/ nano CuO reaction. No evidence of both elemental Al and Cu was found due to structural analysis in the final products. On the other hand, energy dispersive X-ray spectroscopy (EDS) results confirm the appearance of Al and Cu in the produced microstructure. Further investigation may generate belief that Al and Cu ingress within TiC structure causing a fluctuation in its measured lattice constant.

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