Background: Preventing postoperative atelectasis in surgical patients with impaired pulmonary function is preferable. This study presented a case in which the use of nasal high flow treatment to assist the respiratory system prevented the recurrence of postoperative atelectasis. Case Presentation: An urgent laparoscopic procedure was performed on a 68-year-old man who had a perforation in his upper digestive tract. The patient appeared undernourished because of his severe, uncontrolled diabetes mellitus. The scheduled surgery was completed without incident. On the fifth postoperative day, the patient exhibited acute atelectasis of the left lower lobe, with an oxygen saturation of 89%. When tracheal suction and postural draining restored normal oxygenation, positive-pressure ventilation at 8 cm H2O airway pressure was employed to stop recurring atelectasis. On the seventh postoperative day, 15 hours after positive pressure breathing ended, the patient suffered a relapse of acute atelectasis. Once bronchoscopy was utilized to remove a mucous plaque from the tracheobronchial tree, nasal high flow treatment at a rate of 40 liters per minute was employed to deliver a modest dose of continuous positive airway pressure rather than positive pressure breathing. The respiratory rehabilitation continued without a hitch, and his health was more stable with nasal high flow therapy's breathing support than it was with positive pressure ventilation. The nasal high flow therapy also provided gas that was sufficiently humidified, which improved the bronchial secretion. There was no new atelectasis throughout the remainder of the patient's stay in the intensive care unit. Conclusion: Respiratory support with nasal high flow treatment might help to prevent postoperative atelectasis by providing continuous positive airway pressure in conjunction with progressive respiratory rehabilitation.

Background: It is well recognized that manual chest compression could cause a number of internal injuries, many of which are fatal. Similar to this, many type A acute aortic dissection patients who develop cardiac tamponade die before reaching the hospital. This study presented a case in which pericardial laceration brought on by chest compressions have unintentionally caused cardiac tamponade by acute aortic dissection. Case presentation: A 65 year old woman with a history of systemic hypertension was presented who was fainted shortly after complaining of terrible epigastric discomfort. Paramedics started manual chest compressions on the patient, as soon as they realized they were dealing with a cardiopulmonary arrest. The pericardial laceration brought on by chest compressions might have unintentionally freed cardiac tamponade caused by acute aortic dissection, perhaps sparing the patient's life. During surgery, a hemothorax and pericardial laceration connected to the left pleural space were found, but no heart injury was seen. There were no indications of the ruptured aortic aneurysm or any intra-thoracic injuries that would have caused the hemothorax. Hemiarch replacement was successfully accomplished, but the patient died from multi-organ failure 31 days after the operation. Conclusion: An instance of non-fractured pericardial injury caused by chest compression was presented. The pericardial laceration, also afforded the patient enough time for surgery, have accidentally eased the hemothorax brought on by the acute aortic dissection.