AR Scorpii is an intermediate polar system composed of a magnetic white dwarf (WD) and an M-type star, and shows non-thermal, pulsed, and highly linearly polarized emission. The radio/optical emission modulates with the WD's spin and show the double peak structure in the light curves. In this paper, we discuss a possible scenario for the radiation mechanism of AR Scorpii. The magnetic interaction on the surface of the companion star produces an outflow from the companion star, the heating of the companion star surface, and the acceleration of electrons to a relativistic energy. The accelerated electrons, whose typical Lorentz factor is ∼ 50 − 100, from the companion star move along the magnetic field lines toward the WD surface. The electrons injected with the pitch angle of sin θ p,0 > 0.05 are subject to the magnetic mirror effect and are trapped in the closed magnetic field line region.We find that the emission from the first magnetic mirror points mainly contributes to the observed pulsed emission and the formation of the double-peak structure in the light curve. For the inclined rotator, the pulse peak in the calculated light curve shifts the position in the spin phase, and a Fourier analysis exhibits a beat frequency feature, which are consistent with the optical/UV observations. The pulse profile also evolves with the orbital phase owing to the effect of the viewing geometry. The model also interprets the global features of the observed spectral energy distribution in radio to X-ray energy bands. We also discuss the curvature radiation and the inverse-Compton scattering process in the outer gap accelerator of the WD in AR Scorpii and discuss the possibility of the detection by future high-energy missions.
Background
The association between Helicobacter pylori (H. pylori) infection and nonalcoholic fatty liver disease (NAFLD) has been shown in many observational studies, but these conclusions remain controversial. Hence, we performed a meta‐analysis to elucidate the association.
Methods
A comprehensive search was conducted on relevant studies published from inception to December 31, 2018, in PubMed, EMBASE, and Web of Science databases. Odds ratio (OR) with 95% confidence interval (95% CI) were pooled by random‐effect model, generic inverse variance method. Subgroup and sensitivity analyses were also done. Publication bias was estimated by the funnel plot, Begg's test, and Egger's test.
Results
Fifteen studies (eleven cross‐sectional, two case‐control, and two cohort studies) were included in this meta‐analysis. The pooled OR of NAFLD in patients with H. pylori infection was 1.19 (95% CI: 1.11‐1.29, P < 0.00001) when compared with the patients without H. pylori infection. Similar results were observed when the subgroup analyses were stratified by different geographical locations, study designs, and confounders adjustment. In subgroup analysis stratified by different H. pylori testing methods, the correlation still exists when using UBT, serology, RUT, or SAT, but there was no statistically significant difference when using multiple detection methods (OR = 2.96, 95% CI: 0.37‐23.94, P = 0.31). Sensitivity analyses showed that our results were robust. No evidence of substantial publication bias was detected.
Conclusions
Current evidence indicated that a positive association between H. pylori infection and the risk of NAFLD. Further prospective studies are warranted to strengthen the association and to clarify whether there is a causative link between them.
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