The present paper describes a family in which a mother and son are affected with microtia and meatal atresia. The pedigree suggests autosomal dominant inheritance.

We report a brother and sister with congenital chylous ascites. Parents were first cousins, and autosomal recessive inheritance may be the cause of the condition in these sibs.

Concanavalin A and wheat germ agglutinin, lectins that interact with serum glycoprotein in a manner similar to the antigen--antibody reaction, were used as "antibodies" in a single radial immunodiffusion technique to test a coded serum panel (from the National Cancer Institute, Bethesda, Md., and the Mayo Clinic, Rochester, Minn.) containing a) 99 serum samples from patients with different types of malignant neoplasms of the gastrointestinal tract, prostate gland, and lung, b) 50 samples from patients with benign diseases of the same organs as those affected in the cancer patients, and c) 50 samples from apparently healthy smokers. The resulting precipitation rings were not correlated to serum protein concentration, and the differences (demonstrated by Student's t-test and with a generalization of the one-sided two-sample Kolmogorov-Smirnov statistic for evaluating diagnostic tests) established that serum glycoproteins are glycosylated differently in cancer patients than in people without cancer.

We describe 2 sibs; brother and sister, with corneal dermoids and proportionately short stature. It is suggested that this is an autosomal recessive condition.

This report describes a probably new syndrome seen in a brother and sister and characterized by joint laxity, pectus carinatum and a peculiar face with mild frontal bossing, low nasal bridge, lateral displacement of the inner canthi, malar hypoplasia, parrot‐like nose, chubby cheeks, striking philtrum and arched upper lips.