Introduction:The Neurological injury is an important complication after CABG. There is considerable evidence that early postoperative cognitive dysfunction is related to a combination of three factors often associated with CPB: (micro) embolism, hypoperfusion, and the systemic inflammatory response. Intraoperative formation of gaseous emboli and aggregated platelets, atherosclerotic debris, hypoperfusion, hypotension, hyperthermia, hyperglycaemia, surgical trauma, blood loss, Anaesthetic agents, arrhythmia and transfusion all enhance the risk of cognitive dysfunction.There are two types of Neurological injury, Type-I includes stroke, transient ischemic attack and coma and the incidence is approximately 3 to 6 percent. Type-II injury is more subtle and includes impairment of cognitive functions. These defects associated with attention, concentration, short term memory, fine motor function and speed of the mental response. The incidence of neurocognitive impairment after cardiac surgery varies from 20% to 80%. 1 Based on prospective studies, however it is apparent that the incidence of subtle postoperative neurologic and neuropsychological abnormalities is much higher, closer to 50 percent in the first week after cardiac surgery. 2 These apparent high rate of subtle neurologic impairment detected prospectively are in sharp contrast to the considerably lower incidence of stroke after cardiac surgery, reported as 1-5 percent, in several large retrospective series from different centers. 3 There are several reasons for these apparent differences in the reported incidences the timing, thoroughness and the reproducibility of the neurologic examinations, as well as the incorporation of the preoperative assessment for comparison, all determine the sensitivity and Evaluation of Neurocognitive Dysfunction afterCoronary Artery Bypass Surgery p = 0.002 and 25.93 ± 3.11 vs. 26.63 ± 2.50, p = 0.023 respectively