The development and validation of a new measure, the Eating Disorder Inventory (EDI) is described. The EDI is a 64 item, self‐report, multiscale measure designed for the assessment of psychological and behavioral traits common in anorexia nervosa (AN) and bulimia. The EDI consists of eight sub‐scales measuring: 1) Drive for Thinness, 2) Bulimia, 3) Body Dissatisfaction, 4) Ineffectiveness, 5) Perfectionism, 6) Interpersonal Distrust, 7) Interoceptive Awareness and 8) Maturity Fears. Reliability (internal consistency) is established for all subscales and several indices of validity are presented. First, AN patients (N = 113) are differentiated from female comparison (FC) subjects (N = 577) using a cross‐validation procedure. Secondly, patient self‐report subscale scores agree with clinician ratings of subscale traits. Thirdly, clinically recovered AN patients score similarly to FCs on all subscales. Finally, convergent and discriminate validity are established for subscales. The EDI was also administered to groups of normal weight bulimic women, obese, and normal weight but formerly obese women, as well as a male comparison group. Group differences are reported and the potential utility of the EDI is discussed.

This article examines the measurement of short-lived (i.e., state) changes in self-esteem. A new scale is introduced that is sensitive to manipulations designed to temporarily alter self-esteem, and 5 studies are presented that support the scale's validity. The State Self-Esteem Scale (SSES) consists of 20 items modified from the widely used Janis-Field Feelings of Inadequacy Scale (Janis & Field, 1959). Psychometric analyses revealed that the SSES has 3 correlated factors: performance, social, and appearance self-esteem. Effects of naturally occurring and laboratory failure and of clinical treatment on SSES scores were examined; it was concluded that the SSES is sensitive to these sorts of manipulations. The scale has many potential uses, which include serving as a valid manipulation check index, measuring clinical change in self-esteem, and untangling the confounded relation between mood and self-esteem.

We first establish the association between binge eating and dieting and present sequence data indicating that dieting usually precedes binging, chronologically. We propose that dieting causes binging by promoting the adoption of a cognitively regulated eating style, which is necessary if the physiological defense of body weight is to be overcome. The defense of body weight entails various metabolic adjustments that assist energy conservation, but the behavioral reaction of binge eating is best understood in cognitive, not physiological, terms. By supplanting physiological regulatory controls with cognitive controis, dieting makes the dieter vulnerable to disinhibition and consequent overeating. Implications for therapy are discussed, as are the societal consequences of regarding dieting as a "solution" to the problem of binging.

Anorexia nervosa and bulimia nervosa have emerged as the predominant eating disorders. We review the recent research evidence pertaining to the development of these disorders, including sociocultural factors (e.g., media and peer influences), family factors (e.g., enmeshment and criticism), negative affect, low self-esteem, and body dissatisfaction. Also reviewed are cognitive and biological aspects of eating disorders. Some contributory factors appear to be necessary for the appearance of eating disorders, but none is sufficient. Eating disorders may represent a way of coping with problems of identity and personal control.

It was hypothesized that individual differences in eating behavior based on the distinction between obese and normal subjects could be demonstrated within a population of normal subjects classified as to the extent of restraint chronically exercised with respect to eating. Restrained subjects resembled the obese behaviorally, and unrestrained subjects resembled normals. This demonstration was effected in the context of a test .of the psychosomatic hypothesis of obesity. The results indicated that although some individuals may eat more when anxious, there is little empirical support for the notion that eating serves to reduce anxiety. An explanation for this apparent inconsistency was offered.