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An optimal size of postembryonic root apical meristem (RAM) is achieved by a balance between cell division and differentiation. Despite extensive research, molecular mechanisms underlying the coordination of cell division and differentiation are still fragmentary. Here, we report that ORESARA 15 (ORE15), an Arabidopsis PLATZ transcription factor preferentially expressed in the RAM, determines RAM size. The primary root length, RAM size, cell division rate, and stem cell niche activity were reduced in an ore15 loss-of-function mutant but enhanced in an activation-tagged line overexpressing ORE15 compared with the wild type. ORE15 forms mutually positive and negative feedback loops with auxin and cytokinin signaling, respectively. Collectively, our findings imply that ORE15 controls the RAM size by mediating the antagonistic interaction between auxin and cytokinin signaling-related pathways.

This paper proposes a new amorphous indium gallium zinc oxide (a‐IGZO) pixel circuit for the simultaneous emission active‐matrix organic light‐emitting diode (AMOLED) displays, which can compensate for the non‐uniform threshold voltage (VTH) and mobility (μ) of the thin‐film transistor (TFT). The Negative VTH are successfully compensated by adopting the source follower method, and then mobility compensation is carried out by additional voltage adjustment. The OLED current error rate is within 9 % against the VTH and mobility variations in the simulation.

This paper proposes a compensation pixel circuit for stretchable active‐matrix organic light‐emitting diode (AMOLED) display that compensates for the threshold voltage variation and luminance reduction caused by display stretching. The simulated results of the current error rate are suppressed under 10 % at 20 % strain with ±0.5 V threshold voltage shift. Also, the luminance variation was significantly reduced from 19.3 % to 0.12 %.

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