Conversion of the greenhouse gas CO2 to value‐added products is an important challenge for sustainable energy research. Here, a durably nanohybrid composed of Ag nanoparticles and polyacrylamide was constructed for the selectively electroreduction of CO2 to CO. The nanohybrid exhibited an outstanding CO faradaic efficiency of 97.2±0.2 % at −0.89 VRHE (vs. the reversible hydrogen electrode) with a desirable CO partial current density of −22.0±2.3 mA cm−2 and maintained the CO faradaic efficiency above 95 % over a wide potential range (−0.79 to −1.09 VRHE), showing excellent stability during a 48 h prolonged electrolysis. The origins of selective enhancement of CO2 reduction over the nanohybrid stemmed from the activation of CO2 via hydrogen bond and the low basicity of the amide. DFT calculations implied that the synergy of Ag nanoparticles and amide could better stabilize the key intermediate (*COOH) and effectively lower the overpotential of CO2 reduction. These results establish the synergistic effects of organic/inorganic hybrid as a complementary method for tuning selectivity in CO2‐to‐fuels catalysis.

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Green tea has significant protective activity on nerve cells, but the mechanism of action is unclear. Epigallocatechin gallate (EGCG) and N-ethyl-L-glutamine (L-theanine) are the representative functional components of green tea (Camellia sinensis). In this study, an AD model of Aβ25–35-induced differentiated neural cell line PC12 cells was established to study the synergistic effect of EGCG and L-theanine in protecting neural cells. The results showed that under Aβ25–35 stress conditions, mitochondria and axons degenerated, and the expression of cyclins was up-regulated, showing the gene and protein characteristics of cellular hyperfunction. EGCG + L-theanine inhibited inflammation and aggregate formation pathways, significantly increased the percentage of G0/G1 in the cell cycle, downregulated the expression of proteins such as p-mTOR, Cyclin D1, and Cyclin B1, upregulated the expression of GAP43, Klotho, p-AMPK, and other proteins, promoted mitochondrial activity and energy metabolism, and had repair and regeneration effects on differentiated nerve cells. The synergistic mechanism study showed that under the premise that EGCG inhibits amyloid stress and inflammation and promotes metabolism, L-theanine could play a nourish nerve effect. EGCG + L-theanine keeps differentiated nerve cells in a quiescent state, which is beneficial to the repair and regeneration of nerve cells. In addition, EGCG + L-theanine maintains the high-fidelity structure of cellular proteins. This study revealed for the first time that the synergistic effect of EGCG with L-theanine may be an effective way to promote nerve cell repair and regeneration and slow down the progression of AD. Our findings provide a new scientific basis for the relationship between tea drinking and brain protection.

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