A herpes-like virus has been isolated from fetal and neonatal dogs that died with an acute hemorrhagic disease. The virus differs from the other herpes-like viruses in its immunologic and growth characteristics. It is capable of passing the placental barrier and of causing disease and death of some of the fetuses, but in other apparently healthy animals the virus may remain latent, becoming activated under certain conditions.

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Several cell lines that were derived from primates and inoculated with virus originally obtained from a spontaneous mammary carcinoma showed cytopathic effects characterized by multinucleation. These cytopathic effects appeared as early as 24 holurs after inoculation. Multinucleated cells contained virus particles characteristic of the original virus isolate.