Our results suggest that proliferation in advanced human carotid lesions is confined to the intima and focally concentrated in central plaque regions negative for alpha-actin. Furthermore, it apparently occurs primarily as part of inflammatory processes and structural repair predominantly involving macrophages, as well as unspecific mesenchymal cells.

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AbstractThe ethenedithiolato complex [(CO)3FeSCH2]2 (1) is obtained by thermal fragmentation of thiirane-S-oxide in the presence of Fe3(CO)12 in THF and by photolytic reaction of [(CO)3FeS]2 (3) with ethene, respectively. According to an X-ray structure analysis, complex 1 has the same symmetric polyhedron structure as dihydrobenzvalene 2 .