Summary A series of experiments was performed in an attempt to establish the anatomical localization of the effects upon uterine movements and ovulation due to the stimulation of the amygdaloid complex. A remarkable change in the uterine movements was recognized through electrical or chemical stimulation of delimitd parts of the amygdaloid nuclei in dogs, cats and rabbits. In pxtpartum and in non‐pregnant matured animals, as well as in the latter half of pregnancy, an increase of tonus in uterine musculature and auginentation in frequency and amplitude of uterine motility can be evoked through unilateral stimulation of the medial principal amygdaloid nucleus (T of Völsch). These effects vary more or less according to the animal species and rather greatly by the stadium of sexual cycle at the time of experimentation. Stimulation of the lateral principal nucleus (M of VöZsch), the intermediate principal nucleus (T' of Vösch), the so‐called cortical amygdaloid nucleus (B of Vösch), the medial amygdaloid nucleus (D of Vösch) and the central amygdaloid nucleus (E of Vösch) seems to be altogether ineffective. Stimulation of the periamygdaloid cortex gives an equally hardly effect upon the uterine motility. The impulse from the amygdala passes very likely through fibers corresponding to MV or MD fiber described by Fukuchi in Ungulata or through so‐called medial forebrain bundle and is led to the viscero effective centre of the hypothalamus. The electrical stimulation of nuclei T and T' of the amygdala is capable of producing ovulation or hemorrhagic follicle in the ovary of the matured estrous rabbit. The impulse seems to go from the amygdala directly to the ventro medial nucleus of the hypothalamus leading further to the anterior pituitary lobe. The further effect to the ovary may be governed by the gonado tropic hormone released from the anterior pituitary lobe. In this latter case the estrous phase of the animals may also play an important rôle. (This work was aided in part by a grant from the Science Research Foundation of the Educational Department.