The present study showed that whole blood passage time is shorter in females than in males for young people. This conforms to the pattern shown in previous studies which investigated blood passage time among the elderly and people in their prime of life. It is conceivable that females have a higher fluidity than males in all age brackets. Regarding the effects of lifestyle on hemorheology, the present study suggests that several lifestyle factors are related to whole blood passage time and their effects differ according to gender.

No abstract

No abstract

Patient: Female, 49Final Diagnosis: Cerebral venous thrombosisSymptoms: Altered mental state • weakness in limbsMedication: —Clinical Procedure: —Specialty: Endocrinology and MetabolicObjective:Rare co-existance of disease or pathologyBackground:Cerebral venous thrombosis (CVT) is a rare but fatal complication of hyperthyroidism that is induced by the hypercoagulable state of thyrotoxicosis. Although it is frequently difficult to diagnose CVT promptly, it is important to consider it in the differential diagnosis when a hyperthyroid patient presents with atypical neurologic symptoms.Care Report:A 49-year-old Japanese female with unremarkable medical history came in with thyroid storm and multiple progressive ischemic stroke identified at another hospital. Treatment for thyroid storm with beta-blocker, glucocorticoid, and potassium iodide-iodine was started and MR venography was performed on hospital day 3 for further evaluation of her progressive ischemic stroke. The MRI showed CVT, and anticoagulation therapy, in addition to the anti-thyroid agents, was initiated. The patient’s thyroid function was successfully stabilized by hospital day 10 and further progression of CVT was prevented.Conclusions:Physicians should consider CVT when a patient presents with atypical course of stroke or with atypical MRI findings such as high intensity area in apparent diffusion coefficient (ADC) mapping. Not only is an early diagnosis and initiation of anticoagulation important, but identifying and treating the underlying disease is essential to avoid the progression of CVT.

Stanford type A acute aortic dissection (AAD) is associated with carotid artery dissections (CADs). We report two cases of carotid artery stenting (CAS) for symptomatic CAD after ascending aortic replacement (AAR) for AAD.Case Presentation: Case 1: A 51-year-old man with AAD was transferred to our institute. He had no notable paralysis symptoms on initial presentation. However, after AAR for AAD was performed, left paralysis developed within a few hours.Emergency angiography revealed right CAD and pseudo-occlusion. CAS was performed successfully using intravascular ultrasound (IVUS). He was transferred to a rehabilitation hospital with a modified Rankin Scale (mRS) score of 2.Case 2: A 55-year-old man underwent AAR for AAD, but asymptomatic left CAD remained. Two weeks after the operation, he presented with slight signs of aphasia. Aspirin was prescribed and follow-up was performed, but his symptoms did not improve.He underwent magnetic resonance imaging in our department, which revealed acute cerebral infarction on the left pars opercularis and an artery-to-artery embolism from CAD. CAS was performed via the retrograde approach with direct puncture of the normal left common carotid artery using IVUS. He was discharged with no complications and a mRS score of 1. Conclusion:IVUS can be useful for CAS to confirm the true lumen and extension of long CAD lesions developing from AAD.Keywords▶ carotid artery stenting, carotid artery dissection, Stanford type A aortic dissection, intravascular ultrasound Case Presentation Case 1Patient: A 51-year-old man. Complaint: Left hemiplegia. Medical history: Hypertension. Present illness: Suddenly, dimmed vision and weakness were developed and he was transported to his previous hospital by ambulance. On arrival, consciousness was clear and there was no obvious limb paralysis on gross motor movement. The blood pressure was 132/88 mmHg and electrocardiography demonstrated no abnormality. However, detailed examination led to a diagnosis of AAD. The patient was referred to our hospital and emergency AAR was performed at the Department of Cardiovascular Surgery. Immediately after surgery, there was no paralysis of the limbs, but incomplete paralysis of the left upper and lower limbs was suspected 7 hours after surgery, leading to complete paralysis 9 hours after surgery. The patient was referred to the Department of Neurosurgery. No acutephase lesion was found on computed tomography (CT). Three-dimensional (3D) CT angiography (3D-CTA) revealed CAD involving the origin of the brachiocephalic This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives International License.