Traumatic avulsion of the triceps tendon was diagnosed in a 15-month-old, male, neutered European shorthair cat. Diagnosis was established clinically by palpation of a transverse groove proximal to the olecranon and by radiography. The avulsed end of the tendon was surgically reapposed using a modified three-loop pulley suture and horizontal mattress sutures. Postoperatively, elbow flexion was limited for three weeks with the aid of a spica splint and by exercise restriction for six weeks. The cat showed no lameness after bandage removal up to the time of writing (seven months). Although rare, triceps tendon injuries can occur after a blunt trauma and should be included in the differential diagnosis of foreleg lameness in the cat. The modified three-loop pulley suture in combination with subsequent immobilisation of the limb with a splinted bandage resulted in a successful outcome in this cat with a triceps tendon avulsion.

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Two cats with intractable idiopathic chylothorax and a history of unsuccessful medical management were treated thoracoscopically with en bloc thoracic duct sealing and subtotal pericardectomy using a bipolar feedbackcontrolled vessel sealing device. No surgical complications were observed. Twenty-four and 26 months after surgery, both cats were free of thoracic effusion and clinical signs.

This report describes the occurrence of non-weightbearing lameness caused by Mycoplasma felis monoarthritis in two, immunocompetent, European, shorthair adult cats with a suspected history of trauma. Clinical signs recurred after conservative treatment. The joints were treated surgically and M felis was identified as the causative agent for the monoarthritis. Medication with 10 mg/kg doxycycline twice daily was initiated according to susceptibility testing. One cat underwent further joint flushing after two weeks; both the cats recovered completely after eight and nine weeks, respectively. The findings suggest that M felis, in addition to being an agent associated with conjunctivitis in cats, is able to act as a pathogen in other tissues and cause arthritis even in immunocompetent cats.

Pancreatic trauma and rupture are rare after feline high-rise syndrome; however, should it happen, pancreatic enzymes will leak into the abdominal cavity and may cause pancreatic autodigestion and fatty tissue saponification. If not diagnosed and treated, it can ultimately lead to multiorgan failure and death. In this case series, 700 records of high-rise syndrome cats that presented between April 2001 and May 2006 were analysed, and four cats with pancreatic rupture were identified. Clinical signs, diagnosis using ultrasonography and lipase activity in blood and abdominal effusion, and treatment modalities are reported. Three cats underwent surgical abdominal exploration, one cat was euthanased. Rupture of the left pancreatic limb was confirmed in all cases. Two of the operated cats survived to date. High-rise syndrome can lead to abdominal trauma, including pancreatic rupture. A prompt diagnosis and surgical treatment should be considered.