According to theory, maternal sensitivity should be associated with attachment security in middle childhood. We measure two aspects of maternal sensitivity— affective understanding, a component of parental mentalization, and affective synchrony, a component of parental empathy. We tested our hypotheses within a diverse sample of school-aged children (48.6% female, Mage = 10.27, SDage = 1.09) and their mothers ( N = 112 dyads) at baseline and after a standardized laboratory-based stressor in which children worked on unsolvable puzzles while their mothers watched. Results revealed no significant associations at baseline, but lower maternal attachment avoidance and greater child attachment security were associated with greater affective understanding and greater affective synchrony after the stressor task.

The development of strategies that support autonomous self-regulation of emotion is key for early childhood emotion regulation. Children are thought to transition from predominant reliance on more automatic or interpersonal strategies to reliance on more effortful, autonomous strategies as they develop cognitive skills that can be recruited for self-regulation. However, there are few longitudinal studies documenting age-related changes in different forms and dimensions of strategies. The current study tested predicted agerelated changes in strategy use in a task requiring children to wait for something they want. Specifically, we examined the longitudinal trajectories of 3 strategies commonly observed in delayed reward tasks: selfsoothing, seeking attention about the demands of waiting (bids), and distracting oneself. We followed a sample of 120 children (54% male, 93.3% white, from semirural and rural economically strained households) from ages 24 months to 5 years who participated in a waiting task each year. Using growth curve modeling, we found declines in self-soothing, rises and then declines in bidding, and increases in distraction from 24 months to 5 years. Next, we investigated whether strategy use trajectories predicted adult ratings of children's emotion regulation during the task, that is, whether children appeared calm and acted appropriately while waiting. Growth in duration and dominance of distraction use predicted judgments that children were well-regulated by age 5 years, whereas growth in dominance of bidding use negatively predicted being rated as well-regulated. We discuss implications for the understanding of strategy development and future directions, including understanding strategy effectiveness.

Children of mothers with elevated depressive symptoms may observe and learn a maladaptive cognitive style, including low perceptions of agency, that is, low perceived control over their emotions and circumstances. In turn, children may face increased cognitive vulnerability to depressive symptoms; however, this mediational model has yet to be tested. Using a longitudinal design and testing our hypotheses within a community sample, we investigated the mediating role of maternal agency in the associations between maternal depressive symptoms and child behavioral helplessness and depressive symptoms one and a half years later (M ϭ 18.58 months, SD ϭ 6.91 months). A diverse sample of school-age children (N ϭ 137, 57.4% female, M age ϭ 9.74 years, SD age ϭ 1.51 years) and their mothers (N ϭ 122) reported on their depressive symptoms at Time 1. A subsample of children and mothers returned to report on depressive symptoms at Time 2 (N ϭ 68 dyads; 49 with complete data prior to multiple imputation). Maternal agency was coded from narrative responses to the Parent Development Interview-Revised for Parents of School-aged Children (Slade et al., 2009), completed at Time 1. Child behavioral helplessness was indicated by low strategy use at the end of a challenging puzzle task at Time 2. Results revealed that lower maternal agency predicted higher child depressive symptoms at Time 2 and mediated the associations between higher maternal depressive symptoms at Time 1 and higher child behavioral helplessness and depressive symptoms at Time 2. Implications for clinical intervention and future investigations are discussed.