There was no statistical difference in SFCT, but CVI was significantly lower in patients with AMD. Choroidal vascularity index (CVI) was also lower in 'normal fellow' AMD eyes as compared to controls. This suggests possible reduction in choroidal vascularity in eyes with AMD and also to a certain extent in the 'normal fellow' eyes without phenotypical manifestations and may suggest underlying choroidal morphological change leading to wet AMD.

Eye globe abnormalities can be readily detected on dedicated and non-dedicated CT and MR studies. A primary understanding of the globe anatomy is key to characterising both traumatic and non-traumatic globe abnormalities. The globe consists of three primary layers: the sclera (outer), uvea (middle), and retina (inner layer). The various pathological processes involving these layers are highlighted using case examples with fundoscopic correlation where appropriate. In the emergent setting, trauma can result in hemorrhage, retinal/choroidal detachment and globe rupture. Neoplasms and inflammatory/infective processes predominantly occur in the vascular middle layer. The radiologist has an important role in primary diagnosis contributing to appropriate ophthalmology referral, thereby preventing devastating consequences such as vision loss.

Serial measurements of ONH parameters using the Cirrus OCT are found to have good repeatability. The ONH parameters with Cirrus OCT also maintain good repeatability despite head tilt.

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Purpose To evaluate the role of vitrectomy for eyes with dense VH presumed secondary to CNV, as well as their clinical outcomes. Methods Retrospective, consecutive case series from a single centre of 11 eyes of 11 patients over 2 years who underwent vitrectomy, and with no other identified ocular pathology e.g. diabetic retinopathy or trauma. Results Visual acuity (VA) improved in 9 (82%) patients with mean logMAR VA change of 1.19 (+/‐ 0.69). Prior to vitrectomy, intravitreal anti‐vascular endothelial growth factor (anti‐VEGF) had been given to 2 patients; pneumatic displacement of submacular haemorrhage to 1 patient; and photodynamic therapy with verteporfin to 1 patient. There were no pre‐existing retinal breaks found in all patients intraoperatively. Intraoperative complications include 1 posterior capsule rupture with anterior chamber intraocular lens insertion and 2 iatrogenic retinal breaks treated with laser. Angiography identified polypoidal choroidal vasculopathy in six, occult CNV in one and retinal pigment epithelium rip but no identifiable vascular lesion in one. Angiography was not done in three due to extensive disciform scarring. Lesions beyond the vascular arcade were found to have better prognosis. Recurrence of vitreous haemorrhage occurred in 2 patients. Conclusion Vitrectomy for patients with dense VH and presumed CNV (most commonly PCV) was found to be useful to improve visual acuity, especially for lesions beyond the vascular arcade, although effect is limited. Vitrectomy also allows for subsequent retinal evaluation and angiography. The absence of pre‐existing retinal tears is in agreement with the reported outcome that the pathophysiology of breakthrough VH does not involve retinal tears.