Peoples' perception of the necessity of their involuntary admissions is not stable over time and risk of involuntary readmission is associated with recovery style.

Background: Attention Deficit/Hyperactivity Disorder (ADHD) is overrepresented in prison, making

IntroductionThis study explored the reflective writing (RW) of senior medical students across a co-ordinated reflection education programme in General Practice, Paediatrics and Psychiatry clerkships during their transition to clinical clerkships. The study compared RW themes from within and across three clerkships in order to understand the influence clerkships had on experiential learning and developing professional identity. MethodsAll medical students in their penultimate year were invited to participate in the study. 135 reflection assignments were analysed. A qualitative thematic analysis of students' RW was performed. An inductive approach was used and data saturation was achieved.

SummaryAutism spectrum disorders (ASDs) are lifelong conditions. Although not all adults with ASD require psychiatric input, general adult psychiatrists increasingly find themselves responsible for the care of adults with the disorder. This may present a new and unique challenge to them. Here, we summarise the core clinical features of ASD; discuss appropriate diagnostic practice; review the principles of management; and identify key educational, social care and voluntary services for adults with ASD in the UK.

Purpose of Review: Individuals with 22q11.2 Deletion Syndrome (22q11.2DS) have high rates of comorbid mental illness, particularly psychosis and schizophrenia. The purpose of this review is to summarise recent research in the area of 22q11.2DS and psychosis.Recent Findings: Research over the past year has identified negative symptoms, functional impairment, dysphoric mood and a childhood diagnosis of attention deficit hyperactivity disorder as important clinical predictors of psychosis risk in 22q11.2DS. As previously reported in nondeleted schizophrenia, recent studies have implicated neuroinflammation as a possible neurobiological mechanism for psychosis in 22q11.2DS. Recent neuroimaging findings suggest the cortex is significantly thinner in those with 22q11.2DS and psychosis compared to those without psychosis, replicating similar findings in non-deleted schizophrenia. Further data from the International 22q11.2DS Brain and Behavior Consortium has suggested that chromosomal microdeletions are significantly more likely to involve protein-coding genes and several rare copy number variants are associated with the presence of psychosis in deleted individuals. Summary:There have been several significant recent advances to further characterise the high rates of psychosis in 22q11.2DS, to identify additional clinical predictors of psychosis and to increase our understanding of the neural substrate and genetic aetiology of psychosis in 22q11.2DS.