Green technology innovation has become a breakthrough topic in the coordinated development of economic growth and environmental protection. Although the Internet is likely to become a key driver of transformative environmental change and innovation, studies on the impact of internet infrastructure on green innovation and analyses of the paths are still extremely scarce. Based on a sample of China’s listed companies from 2009 to 2019, this paper treats the Broadband China pilot strategy as a quasi-natural experiment and adopts the time-varying difference-in-differences (DID) model to explore the effect and the transmission path of internet infrastructure construction on green innovation. The study finds that Broadband China significantly promotes green innovation, and the result remains consistent after a series of robustness tests. The transmission path test proves that internet infrastructure construction affects green innovation by improving the degree of informatization, human capital, and internet media reports and by reducing financing constraints. Furthermore, considering the heterogeneity effect, the Broadband China strategy has a greater stimulating effect on state-owned, large-scale, high-tech enterprises; enterprises in low-competition industries; enterprises in growth and mature stages; and enterprises registered in the central and eastern regions. This paper systematically analyzes the effects of internet infrastructure on the green innovation of enterprises based on economic informatics theory, providing new insights for improving internet infrastructure and green innovation in practice.

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Background. Vaspin is an important adipokine that is involved in cardiovascular diseases. This study is aimed at investigating whether vaspin participates in sepsis-induced cardiac injury and explored the possible mechanism. Methods. First, cecal ligation and puncture (CLP) and lipopolysaccharide (LPS) were used to establish a mouse model of sepsis, and cardiac vaspin expression was examined. In addition, after pretreatment with vaspin or phosphate-buffered saline (PBS), wild-type (WT) mice underwent CLP to establish a septic model and received sham as a control. Finally, WT mice and kallikrein 7 (KLK7-/-) mice were underwent CLP with or without vaspin pretreatment. Results. Mice that underwent CLP and were administered LPS exhibited increased vaspin expression in both the heart and serum compared with sham- or saline-treated mice. In CLP mice, pretreatment with vaspin reduced mortality and alleviated the expression of cardiac injury markers and cardiac dysfunction. In addition, vaspin reduced the cardiac levels of CD45+ cells and CD68+ cells, alleviated the cardiac inflammatory response, and reduced cardiomyocyte apoptosis. The protective effects of vaspin on CLP mice were masked by the deletion of KLK7, which was demonstrated to be a downstream signal of vaspin. Conclusions. Vaspin alleviates cardiac inflammation and plays a protective role in sepsis-induced cardiac injury by reducing KLK7 expression.

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