H epatocellular carcinoma (HCC) accounts for the majority of primary liver cancers, which are the fourth leading cause of cancer-related deaths worldwide. The burden of HCC is increasing, mainly due to the rising prevalence of obesity and nonalcoholic fatty liver disease (1). Although surveillance programs have been implemented for at-risk patients (eg, patients with cirrhosis) to detect tumors at early stages and to allow for potentially curative treatment, such patients have high recurrence rates and heterogeneous recurrence-free survival (2). Indeed, HCC has strong genomic, molecular, and histologic heterogeneity (3,4). Several histologic subtypes of HCC associated with clinical and molecular features of different prognostic value have been described (5-7), among which the macrotrabecular-massive (MTM) HCC (MTM-HCC) subgroup has been recently identified (7,8). MTM-HCCs have been shown to be associated with poor survival, high serum a-fetoprotein (AFP) level, and histologic features of aggressiveness, including microvascular and macrovascular invasion and satellite nodules (8). This subtype has been newly included in the fifth edition of the World Health Organization Classification of Tumors (9).Identification of the aggressive HCC subtypes during pretherapeutic work-up may have strong prognostic and therapeutic implications (3). In patients with cirrhosis, the diagnosis of HCC may be performed noninvasively using multiphasic CT or dynamic contrast materialenhanced MRI using the Liver Imaging Reporting and Data System (LI-RADS), which provides standardization for HCC imaging acquisition and terminology and allows for accurate stratification of the probability of HCC and overall malignancy (2,10,11). As pathologic diagnosis is thus not systematically required, the identification

ADC variation observed in CRC metastases following systemic chemotherapy reflects a specific increase in free-molecular diffusion (D), in itself correlated to the degree of metastasis necrosis.

Radiologist sensitivity CTC for detection of polyps ≥ 6 mm in training was the sole independent predictor for subsequent sensitivity in detection of such polyps.

Visceral artery aneurysms are rare but their estimated mortality due to rupture ranges between 25 and 70%. Treatment of visceral artery aneurysm rupture is usually managed by interventional radiology. Specific embolization techniques depend on the location, affected organ, locoregional arterial anatomy, and interventional radiologist skill. The success rate following treatment by interventional radiology is greater than 90%. The main complication is recanalization of the aneurysm, showing the importance of post-therapeutic monitoring, which should preferably be performed using MR imaging.