An exciting new prospect in condensed matter physics is the possibility of realizing fractional quantum Hall states in simple lattice models without a large external magnetic field. A fundamental question is whether qualitatively new states can be realized on the lattice as compared with ordinary fractional quantum Hall states. Here we propose new symmetry-enriched topological states, topological nematic states, which are a dramatic consequence of the interplay between the lattice translational symmetry and topological properties of these fractional Chern insulators. The topological nematic states are realized in a partially filled flat band with a Chern number N, which can be mapped to an N-layer quantum Hall system on a regular lattice. However, in the topological nematic states the lattice dislocations can act as wormholes connecting the different layers and effectively change the topology of the space. Consequently, lattice dislocations become defects with a nontrivial quantum dimension, even when the fractional quantum Hall state being realized is, by itself, Abelian. Our proposal leads to the possibility of realizing the physics of topologically ordered states on high-genus surfaces in the lab even though the sample has only the disk geometry.
It has recently been realized that a general class of non-abelian defects can be created in conventional topological states by introducing extrinsic defects, such as lattice dislocations or superconductor-ferromagnet domain walls in conventional quantum Hall states or topological insulators. In this paper, we begin by placing these defects within the broader conceptual scheme of extrinsic twist defects associated with symmetries of the topological state. We explicitly study several classes of examples, including Z2 and Z3 twist defects, where the topological state with N twist defects can be mapped to a topological state without twist defects on a genus g ∝ N surface. To emphasize this connection we refer to the twist defects as genons. We develop methods to compute the projective non-abelian braiding statistics of the genons, and we find the braiding is given by adiabatic modular transformations, or Dehn twists, of the topological state on the effective genus g surface. We study the relation between this projective braiding statistics and the ordinary nonabelian braiding statistics obtained when the genons become deconfined, finite-energy excitations. We find that the braiding is generally different, in contrast to the Majorana case, which opens the possibility for fundamentally novel behavior. We find situations where the genons have quantum dimension 2 and can be used for universal topological quantum computing (TQC), while the host topological state is by itself non-universal for TQC.
The Lieb-Schultz-Mattis theorem and its higher-dimensional generalizations by Oshikawa and Hastings require that translationally invariant 2D spin systems with a half-integer spin per unit cell must either have a continuum of low energy excitations, spontaneously break some symmetries, or exhibit topological order with anyonic excitations. We establish a connection between these constraints and a remarkably similar set of constraints at the surface of a 3D interacting topological insulator. This, combined with recent work on symmetry-enriched topological phases with on-site unitary symmetries, enables us to develop a framework for understanding the structure of symmetry-enriched topological phases with both translational and on-site unitary symmetries, including the effective theory of symmetry defects. This framework places stringent constraints on the possible types of symmetry fractionalization that can occur in 2D systems whose unit cell contains fractional spin, fractional charge, or a projective representation of the symmetry group. As a concrete application, we determine when a topological phase must possess a "spinon" excitation, even in cases when spin rotational invariance is broken down to a discrete subgroup by the crystal structure. We also describe the phenomena of "anyonic spin-orbit coupling," which may arise from the interplay of translational and on-site symmetries. These include the possibility of on-site symmetry defect branch lines carrying topological charge per unit length and lattice dislocations inducing degeneracies protected by onsite symmetry.
The structure of extrinsic defects in topologically ordered states of matter is host to a rich set of universal physics. Extrinsic defects in 2+1 dimensional topological states include line-like defects, such as boundaries between topologically distinct states, and point-like defects, such as junctions between different line defects. Gapped boundaries in particular can themselves be \it topologically \rm distinct, and the junctions between them can localize topologically protected zero modes, giving rise to topological ground state degeneracies and projective non-Abelian statistics. In this paper, we develop a general theory of point defects and gapped line defects in 2+1 dimensional Abelian topological states. We derive a classification of topologically distinct gapped boundaries in terms of certain maximal subgroups of quasiparticles with mutually bosonic statistics, called Lagrangian subgroups. The junctions between different gapped boundaries provide a general classification of point defects in topological states, including as a special case the twist defects considered in previous works. We derive a general formula for the quantum dimension of these point defects, a general understanding of their localized "parafermion" zero modes, and we define a notion of projective non-Abelian statistics for them. The critical phenomena between topologically distinct gapped boundaries can be understood in terms of a general class of quantum spin chains or, equivalently, "generalized parafermion" chains. This provides a way of realizing exotic 1+1D generalized parafermion conformal field theories in condensed matter systems.Comment: 24 pages, 11 figures. Some of the results here are also summarized in arXiv:1304.7579; v2: strengthened braiding results and mapping to genons, updated refs/acknowledgment
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