We examine the role of ill-health in retirement decisions in Britain, using the first eight waves of the British Household Panel Survey . As self-reported health status is likely to be endogenous to the retirement decision, we instrument self-reported health by a constructed 'health stock' measure using a set of health indicator variables and personal characteristics, as suggested by . Using both linear and non-linear fixed effects estimators, we show that adverse individual health shocks are an important predictor of individual retirement behaviour. We compare the impact of our constructed health measure on economic activity with that arising from the use of other health variables in the data set. We also examine the impact of the 1995 reform of disability benefits on the retirement decision. Key WordsIll health Retirement Disability insurance JEL classification H55 I12 J26 AcknowledgementsWe are grateful to the Nuffield Foundation for funding the project: 'A study of health and the labour market behaviour of older workers', and to Sharon Witherspoon in particular. Executive SummaryThis paper examines the role of ill-health in retirement decisions in Britain, using the first eight waves of the British Household Panel Survey . To tackle the problem that self-reported health status is likely to be endogenous to the retirement decision, a two-stage method, as suggested by , is adopted. The first stage is to construct a 'health stock' measure that is cleansed of the effects of reporting behaviour reflecting labour market participation. This measure is then introduced into a reduced form model of labour market (in)activity. At this latter stage, modelling how labour market transitions are related to time variation in health and other characteristics, helps to eliminate any unchanging person-specific association between characteristics and the decision to work.The 'health-stock' measure is constructed by regressing self-reported health on a set of more objective health indicator variables and a set of other personal characteristics.Few personal characteristics, other than those related to health, are found to be significant in explaining self-assessed health. Many of the health indicators are significant in the model, and as expected having health problems is associated with reporting poor health.The panel structure of the data set is exploited to estimate a non-linear 'fixedeffects' model that allows an exploration of how time variation in various characteristics relates to transitions out of (and in to) work. Deterioration in an individual's health is found to be strongly positively associated with movement out of work. Sensitivity analyses suggest that there may be some asymmetry between the respective effects of health deteriorations and improvements on transitions out of, and in to, work.The final section briefly considers a reform to the public disability insurance programme in 1995, which both tightened formal eligibility conditions and reduced the economic incentives to retire via the disability insu...

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The human X and Y chromosomes share two homologous pseudoautosomal regions (PARs) which pair and recombine at meiosis. PAR1 lies at the tips of the short arms, and the smaller PAR2 at the tips of the long arms. PAR1 contains several active genes, and has been thought to be critical for pairing and fertility. The inconsistent gene content of the PARs between different species of eutherian ('placental') mammals suggests that gene content is immaterial to function, and the failure to detect a PAR at all in some rodents and all marsupials implies that homologous pairing is not universally essential for fertility. The autosomal localization of marsupial homologues of human PAR1 genes and their co-localization with human Xp22 genes implies that the human PAR1 represents a relic of part of an autosomal region added to both X and Y chromosomes between 80 and 130 MYrBP. The same argument may be made for part of PAR2. Independent additions to the sex chromosomes of macropodid marsupials and monotremes can also be inferred from comparative mapping. We conclude that the PARs are relics of differential additions, loss, rearrangement and degradation of the Y chromosome in different mammalian lineages.

This paper uses a realistic structural lifecycle model of consumption and housing decisions to understand how data might distinguish different mechanisms that explain the correlation between house prices and consumption. The model includes price and earnings shocks estimated from data (the latter including aggregate and idiosyncratic components), and incorporates realistic features of the UK mortgage market. We simulate the model using more than 30 years of realized shocks and under counterfactual scenarios. Our results confirm the intuition of earlier studies: house price shocks should have a larger effect on the consumption of older households and earnings shocks on young households.