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Objective: To evaluate the results of subcutaneous anterior transposition of the ulnar nerve in the surgical management of cubital tunnel syndrome and to use FCU aponeurotic fascia to retain the position of anteriorly transposed ulnar nerve. Methods: This is a retrospective study, which evaluated 15 patients (5 males, 10 females; mean age: 48 years; range: 26 to 60 years) who underwent subcutaneous transposition of the ulnar nerve. Mean follow-up period was 2 years (range: 1 year 2 months to 2 years 6 months). Modified McGowan's classification was used for preoperative scoring and the Wilson & Krout classification for postoperative clinical evaluation. Preoperatively 2 patients (13%) had Grade 1, 3 (20%) had Grade 2A, 4 (27%) had Grade 2B, and 6 (40%) had Grade 3 neuropathy. Results: There were excellent results in 10 patients (66%), good in 4 (26%) and fair in 1patient (7%). There was a negative correlation between the preoperative McGowan grade and the postoperative Wilson & Krout score (p<0.05). The success rate of the operation was significantly lower in patient groups as the time from symptom onset increased (p<0.05). There were no complications. Conclusion: Subcutaneous anterior transposition of the ulnar nerve is an effective and reliable surgical method with a low complication rate for the treatment of cubital tunnel syndrome.

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The atrioventricular septal defects encompass a spectrum of lesions in which the common etiology appears to be abnormal development of the superior and inferior endocardial cushions, resulting in a deficiency or absence of the atrioventricular septum [1-6]. This deficiency of the atrioventricular septum results in an ostium primum defect immediately above the atrioventricular valves and a scooped-out area in the inlet (basal) portion of the ventricular septum. Data from the New England Regional Infant Cardiac Program and the Baltimore-Washington infant study defined a prevalence of 0.118 and 0.352 per 1000 live births [7,8]. Approximately, 50-75% of these patients have trisomy 21 [7-9]. When viewed from a reverse perspective, approximately 35-40% of patients with Down’s syndrome have an atrioventricular septal defect [7-9].

The atrioventricular septal defects encompass a spectrum of lesions in which the common etiology appears to be abnormal development of the superior and inferior endocardial cushions, resulting in a deficiency or absence of the the atrioventricular septum [1,2]. Data from the New England Regional Infant Cardiac Program and the Baltimore-Washington infant study defined a prevalence of 0.118 and 0.352 per 1000 live births [3,4]. Approximately, 50-75% of these patients have trisomy 21 [5]. When viewed from a reverse perspective, approximately 35-40% of patients with Down’s syndrome have an atrioventricular septal defect [5].