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Morphometric measurements of the calvarium have demonstrated that the squamous temporal bone is thinner in patients with SCD as compared with controls. Thus, the process leading to defects in the tegmen extends beyond the petrous pyramid. This suggests that there may be extratemporal factors leading to the development of a dehiscence. These findings also have implications for the surgical treatment of this disorder. Resurfacing methods may have a higher failure rate as the bone graft has reduced mass and maybe prone to resorption. Canal plugging methods may provide a more definitive means of addressing the dehiscent labyrinth than resurfacing.

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