[Purpose] The purpose of the study was to investigate the effects of exercise on the recovery of spinal cord nerve cells damaged due to pain signals which are a major symptom of osteoarthritis. [Subjects and Methods] Adult male Sprague-Dawley rats (n=40) were used and induction of osteoarthritis by monosodium iodoacetate. Injected rats were randomly divided into 4 groups: Sham control group without MIA injection (SG), control group with injected MIA (CG), OA without exercise (NEG), OA with exercise (EG). Sham control group was injected normal cell line instead of MIA. The exercise group was submitted to 4-week training program on a treadmill for 5 days/week, 30 min/day, 16 m/min velocity, then spinal cord were removed and measured the GAP-43 expression by immunohistochemistry analysis. [Results] In this study, a results of measuring the expression of GAP-43. GAP-43 was observed in all groups, showed that the significant difference in each group. [Conclusion] It could be seen that exercise increased the GAP-43 expression in the spinal cord to promote the recovery of spinal cord nerve cells damaged due to chronic osteoarthritis.

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METHODS:In this study, 30 subjects were randomly divided into two groups of 15 subjects each, were assigned to the horse riding simulator exercise or the neck exercise group and they performed exercise 30 minutes per each round two times a week for six weeks. To determine the subjects' forward head posture, the three angles and three distances were measured. RESULTS:The forward head angle and head distance results showed a significant change between pre and post intervention in both group. The horizontal distance between acromion and tragus results showed a significant change between pre and post intervention in neck exercise group †Corresponding Author : hbgak@daegu.ac.kr This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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