The purpose of this study was to compare the accuracy of stereolithographic casts (SCs) with those obtained using conventional implant impressions. An epoxy resin model containing dental implants was used as master model. Dental casts (n = 10) were fabricated through both conventional and digital implant impressions. The conventional casts (CCs), SCs, and the master model were digitized, and the accuracy was determined through a deviation analysis and linear measurements. Data were analyzed using paired Student t test with P < .05. The SCs showed higher deviation at the vestibular area (CC: 41 ± 28.87 μm; SC: 117 ± 36.83 μm) and lingual cusps (CC: 40.70 ± 19.79 μm; SC: 80 ± 42.95 μm) in comparison with CCs. No statistically significant difference was found for linear measurements of conventional and digital casts. The entire-arch accuracy was comparable between casts. However, SCs were less accurate at the cusp level in comparison with CCs.
Background. Genetic polymorphisms in certain cytokines and chemokines have been investigated to understand why some individuals display implant flaws despite having few risk factors at the time of implant. Purpose. To investigate the association of genetic polymorphisms in interleukin- (IL-) 10 [-1082 region (A/G)] and RANKL [-438 region (A/G)] with the failure of dental implants. Materials and Methods. This study included 90 partially edentulous male and female patients who were rehabilitated with a total of 245 Straumann dental implants. An implant was considered a failure if any of the following occurred: mobility, persistent subjective complaint, recurrent peri-implant infection with suppuration, continuous radiolucency around the implant, probing depth ≥ 5 mm, and bleeding on probing. Buccal mucosal cells were collected for analysis of RANKL438 and IL-10. Results. The implant success rate in this population was 34.4%. The mutant allele (G) in RANKL had an incidence of 52.3% and mutant allele (A) in IL-10 was observed in 37.8%. No statistically significant difference was detected between the failure of the implant and the genotypes and allelic frequencies. Conclusion. No association was detected between the genetic polymorphisms of RANKL (-438) and IL-10 (-1082) and the failure of dental implants in the population studied.
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