No abstract

Shoulder dislocations are frequently seen in the general population and can be a cause of instability. Instability can lead to debilitating symptoms and morbidity as a result of progressive damage to the shoulder. Anterior shoulder dislocations are the most frequent type of dislocations and have been studied extensively with MRI. The soft tissue Bankart lesion is the most well-known entity associated with anterior instability; however, additional structural lesions arising from traumatic events have been described in recent literature which also predispose to anterior shoulder instability. One of these lesions, the glenoid avulsion of the glenohumeral ligament (GAGL), involves avulsion of the inferior glenohumeral ligament from the glenoid and involves separation from an intact labrum. In contrast to the Bankart lesion, there has been limited discussion of the GAGL lesion in the literature and very few imaging examples. We report a case of a GAGL diagnosed on MRI and confirmed with arthroscopy. It is discussed in the context of the anatomy of the inferior glenohumeral ligament and the imaging findings.

An initial demonstration of minimal-artifact multispectral DWI in the near vicinity of metallic hardware is described and successfully demonstrated on clinical subjects. Magn Reson Med 79:987-993, 2018. © 2017 International Society for Magnetic Resonance in Medicine.

A 32-year-old woman with a previous diagnosis of human immunodeficiency virus infection presented to her primary care provider in 2010 with a progressively enlarging mass in her proximal right forearm. The patient reported a limited range of motion with decreased extension and, to a lesser extent, decreased flexion because of physical obstruction created by the mass. She denied experiencing pain associated with the mass. The patient reported that the area had been injured when she was assaulted with a board several years before. There was no other pertinent medical history. Imaging FindingsRadiography demonstrated a large, lytic lesion causing prominent expansile remodeling of the proximal ulna from the level of the metadiaphysis to the articular surface of the olecranon. The cortex was thin but well formed and intact (Fig 1). The lesion demonstrated trabecular thickening, coarse septa, and several well-circumscribed lucent ovoid foci with scattered peripheral internal regions of calcification but no calcified matrix suggestive of an osteoid or chondroid tumor. There was no adjacent soft-tissue mass.