technique(s) capable of eradicating the cyst stage of T. gondii from chronically infected individuals. Therefore, development of an immunologic intervention capable of attacking and eradicating the cysts is a valuable approach to fight against this widespread infection. Although information exists on the molecular mechanisms of the interferon (IFN)-gemediated protective immunity to control proliferation of T. gondii tachyzoites (the acute stage form), 8,9 the mechanisms of the host immunity against the cyst stage of the parasite are not well understood. Our recent studies revealed that an adoptive transfer of
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