Background: Children are diagnosed as dyslexic when their reading performance is much below that which could be expected for their educational level and cannot be explained by a sensory, neurological or psychiatric deficit or by a low IQ. Although poor reading is a major obstacle to school and career achievement, the causes of dyslexia are unclear and traditional therapies are often unsuccessful. To determine the causes of dyslexia, experiments must demonstrate under which conditions a reading disorder occurs and whether the reading performance improves if these conditions are abolished or compensated. To avoid irreproducible results, experiments must be repeated and the effect size must be calculated. Objectives: The aims of the study were to investigate the rate and location of misread letters within pseudowords, prove the effectiveness of compensatory reading therapy and demonstrate the reproducibility of the experimental results. The influence of reading therapy on the rate of eye movements opposite to the reading direction was investigated and causes of a poor reading performance were identified. Methods: The rate and location of misread letters were investigated by tachystoscopic presentation of pseudowords containing between three and six letters. Presentation time, fixation time, and the time it takes to begin pronouncing the words (speech onset latency) were changed until 95% of the pseudowords were recognized correctly. On the basis of these results, the children learned a reading strategy that compensated the causes of the reading disorder. The therapy was demonstrated to be highly effective and it was shown that the results of the therapy were reproducible. Results: It was shown that misread letters occurred at all locations in pseudowords, regardless of the word´s length. Inadequate fixation, excessively large saccadic amplitudes, reduced ability to simultaneously recognize a sequence of letters, a longer required fixation time and a longer required speech onset latency were all identified as causes of dyslexia. Each of the studies included in the meta-analysis were much more efficient than conventional therapeutic methods. The overall effect size with a value of Hedges´G = 1.72 showed that the therapy had a reproducible and stable effect. Conclusions: The causes of dyslexia can be revealed by a dual-intervention approach consisting of a pseudoword experiment and learning a compensatory reading strategy. Reading performance improves immediately if the identified causes of dyslexia are compensated by an appropriate reading therapy.
Background:Reading disability is termed “dyslexia” if it is much lower than other cognitive abilities according to the intelligence quotient (IQ). This means that dyslexia is caused by an impairment of abilities other than those which the IQ requires. Therefore, reading performance should improve immediately if these impairments are either eliminated or compensated.Objective:The experiments explore conditions under which these impairments are compensated and dyslexic children's poor reading ability immediately improve.Methods:Experiment 1 examined if reducing the number of letters in pseudowords, prolonging the time interval during which the gaze is directed to pseudowords, reducing the amplitude of saccades and prolonging the time interval that elapsed between the beginning of the presentation of a pseudoword and the beginning of the pronunciation of that word influences childrens’ reading performance. A group of 100 German children (71 boys and 29 girls) aged 8 to 13 years, who suffered from dyslexia according to the Zuerich Reading Test, were divided into a training group (n = 50) and an age-matched control group (n = 50) and tested. Both groups participated in experiment 1. Only the children in the training group participated in experiment 2, in which the children learned a compensatory reading strategy. The age - matched control group did not learn the compensatory reading strategy. In the training group, reading performance was tested before and after having learned the new reading strategy.Results:Conditions were found under which all children were able to read 95% of the pseudowords correctly. After having learned a compensatory reading strategy, a mean 58.9% decrease in words read incorrectly was found after a single training session. The difference between the number of reading mistakes before and after the training session was highly significant (Wicoxon Test: p < 0.00001). The effect size showed that the compensatory reading strategy was highly effective (Hedges g = 1.7). The reading ability of an age-matched dyslexic control group showed no improvement.Conclusions:Dyslexic subjects’ reading performance improves significantly when they learn a new reading strategy.
Background: Flawless reading presupposes the ability to simultaneously recognize a sequence of letters, to fixate words at a given location for a given time, to exert eye movements of a given amplitude, and to retrieve phonems rapidly from memory. Poor reading performance may be due to an impairment of at least one of these abilities. Objectives: It was investigated whether reading performance of dyslexic children can be improved by changing the reading strategy without any previous training. Methods: 60 dyslexic German children read a text without and with the help of a computer. A tailored computer program subdivided the text into segments that consisted of no more letters than the children could simultaneously recognize, indicated the location in the segments to which the gaze should be directed, indicated how long the gaze should be directed to each segment, which reading saccades the children should execute, and when the children should pronounce the segments. The computer aided reading was not preceded by any training. Results: It was shown that the rate of reading mistakes dropped immediately by 69.97% if a computer determined the reading process. Computer aided reading reached the highest effect size of Cohen d = 2.649. Conclusions: The results show which abilities are indispensable for reading, that the impairment of at least one of the abilities leads to reading deficiencies that are diagnosed as dyslexia, and that a computer-guided, altered reading strategy immediately reduces the rate of reading mistakes. There was no evidence that dyslexia is due to a lack of eye movement control or reduced visual attention.
This paper investigates whether and to what extent vision with awareness is still possible in the whole visual field after loss of the occipital lobe of one or both cerebral hemispheres or after hemispherectomy in childhood. The visual functions of four children who suffered from unilateral or bilateral loss of the occipital lobe or who had been hemispherectomized were examined. The results show that even after unilateral loss of the striate and prestriate cortex the extent of the visual field may still be in the normal range. The residual visual functions may be mediated by intact extrastriate areas such as V5 and LO of the damaged cerebral hemisphere. It is also shown that even after complete hemispherectomy in early life the visual field may have a normal extent and that conscious visual perception in the whole visual field may be preserved. In hemispherectomized children, the remaining cerebral hemisphere or neural structures in the midbrain, including the superior colliculi and the praetectum, may be able to mediate these visual functions.
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