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In a recent communication Neufeld and associates (1961) described the pathological, functional, electrocardiographic, and clinical findings in a group of eight cases of an uncommon condition in which both great vessels arose from the right ventricle in the absence of pulmonary stenosis. In seven of the eight cases of this condition (for which such synonyms as "partial transposition," "Taussig-Bing complex," "double outlet right ventricle" have been offered) the picture both hmmodynamically and clinically resembled that in large ventricular septal defects.One of the patients (Case 4) of the earlier report, which emanated from the Mayo Clinic, presented somewhat different features from the remaining seven and resembled not only the case reported by Taussig and Bing (1949) but also others reported later as examples of the so-called Taussig-Bing complex. Distinguishing features of the latter type of case included cyanosis and higher levels of oxygen saturation of blood in the pulmonary artery than in a systemic artery or the aorta.This difference in groups of patients having apparently similar anatomical findings (that is, origin of both great vessels from the right ventricle without pulmonary stenosis) prompted a review of another series represented by ten cases from the University of Minnesota Hospitals. From this material a report of correlative studies of the anatomical findings with clinical, hcmodynamic, and angiocardiographic findings represents the main purpose of this communication.These studies suggest the value of classifying into two subdivisions the cases manifesting origin of both great vessels from the right ventricle without pulmonary stenosis. Furthermore, we believe, on the basis of the evidence we shall present, that the so-called Taussig-Bing complex represents one of these two subdivisions. Nine of the ten patients with the anomaly came to necropsy, while the tenth (Case 9), later to be classified as an example of Type IIA, was studied only clinically. The anatomical similarities in all cases of origin of both great vessels from the right ventricle regardless of subdivision are as follows.1. The pulmonary trunk and aorta are normally related externally. 2. The aorta originates entirely from the right ventricle lying to the right of the pulmonary trunk.3. The aortic valve lies higher than normal and is located at about the same cross-sectional and coronal body planes as the pulmonary valve.4. Discontinuity of mitral and aortic valvular tissue is the usual finding (in seven of nine cases in the current study). In the nine examples of the malformation for which necropsy specimens are

Anatomic, clinical, hemodynamic, and roentgenographic findings in six patients with congenital communication of a coronary artery with a cardiac chamber or the pulmonary trunk are presented. The literature is reviewed.A coronary artery may communicate anomalously, with any of the cardiac chambers, more commonly with those on the right side. In the six cases presented, the right coronary artery communicated with the right atrium in one case and with the right ventricle in another. The left coronary artery communicated with the right atrium and right ventricle in one case each, and with the pulmonary trunk in two cases.The most striking feature observed clinically was a continuous murmur. If a continous murmur is localized in an area atypical for patent ductus arteriosus, the diagnosis should be suspected. Conventional roentgenographic and electrocardiographic studies yielded no specific diagnostic features. The results of cardiac catheterization may reveal a left-to-right shunt, but they are diagnostically useful only when correlated with clinical findings.The only precise method of demonstrating the abnormality is by means of selective aortography performed by injecting medium into the very origin of the aorta.Cure is possible by surgical interruption of the fistulous tract.

Computed tomography (CT) and radiography were used to study 20 patients with chest injuries. Excluding rib fractures, CT revealed 50 traumatic lesions ranging from extrapleural hematomas to pericardial fluid collections. In contrast, only 12 lesions were detected on the corresponding radiographs. While many of the processes demonstrated on CT were not life-threatening, substantial abnormalities such as pericardial effusion and diaphragmatic rupture were detected.