Premature autonomy describes a developmental dynamic where parents of high‐risk adolescents reduce their involvement and guidance when confronted with challenges of problem behaviour and the influence of deviant friendships. This dynamic was tested on the sample of Oregon Youth Study boys (N=206), whose family management practices and friendships were observed on videotaped interaction tasks. Latent growth curve models were used to examine longitudinal trends between deviant friendship interactions and family management. Direct observations of deviant friendship process at age 14 were associated with degradation in family management during adolescence. A comparison of antisocial and well‐adjusted boys clarified that parents of antisocial boys (started early and persisted) decreased family management around puberty, in comparison to parents of well‐adjusted boys who maintained high levels of family management through adolescence. In predicting late adolescent problem behaviour, there was a statistically reliable interaction between family management degradation and deviant peer involvement in adolescence in support of the premature autonomy hypothesis. Adolescent males involved in deviant friendships, and whose parents decreased their family management, were most likely to use marijuana and commit antisocial acts at age 18. The implications for interventions that target adolescents are discussed.

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Traditional attribution theory conceptualizes explanations of behavior as referring to either dispositional or situational causes. An alternative approach, the folk-conceptual theory of behavior explanation, distinguishes multiple discrete modes of explanation and specific features within each mode. Because attribution theory and the folk-conceptual theory carve up behavior explanations in distinct ways, they offer very different predictions about actor-observer asymmetries. Six studies, varying in contexts and methodologies, pit the 2 sets of predictions against each other. There was no evidence for the traditional actor-observer hypothesis, but systematic support was found for the actor-observer asymmetries hypothesized by the folk-conceptual theory. The studies also provide initial evidence for the processes that drive each of the asymmetries: impression management goals, general knowledge, and copresence.

Although enhanced sympathetic tone is a well recognized component of the autonomic profile characteristic of congestive heart failure, the contribution of parasympathetic withdrawal to this autonomic imbalance is less well described. The technique of spectral analysis of heart rate variability provides a dynamic map of sympathetic and parasympathetic tone and was thus used to define the nature of sympathetic-parasympathetic interactions in humans with idiopathic dilated cardiomyopathy and in a paced canine model of congestive heart failure. Humans with cardiomyopathy were found to have an augmentation of the sympathetically mediated low frequency area of the power density spectrum. Parasympathetic withdrawal was demonstrated by significant reductions in the parasympathetically mediated high frequency area (p less than 0.05) and the ratio of high to low frequency areas (p less than 0.01). Administration of atropine to normal subjects resulted in a significant reduction in the high frequency area (p less than 0.05) and the high/low frequency area ratio, both of which decreased within the range noted in patients with congestive heart failure. Administration of isoproterenol in normal subjects led to an augmentation of the low frequency area but to only a small decrease in the high/low frequency area ratio. Induction of congestive heart failure in a paced canine model resulted in alterations in the autonomic profile that resembled those seen in humans with ventricular failure. The prominent high frequency region of the spectrum at baseline, indicating a predominance of parasympathetic tone, was absent after the evolution of congestive heart failure, and there was a marked augmentation of the low frequency region of the spectrum.(ABSTRACT TRUNCATED AT 250 WORDS)

Drug abuse and impulsive choice are related in humans. In female rats, impulsive choice predicted the rate of acquisition of IV cocaine self-administration. The objectives of the present experiments were to: (a) compare impulsive choice in males and females, (b) extend previous research on impulsive choice and acquisition of cocaine self-administration to males, and (c) compare males and females during maintenance, extinction, and reinstatement of cocaine-seeking behavior. Male and female rats were trained on an adjusting delay task in which a response on one of two levers yielded one food pellet immediately, and a response on the other resulted in three pellets after an adjusting delay that decreased after responses on the immediate lever and increased after responses on the delay lever. A mean adjusted delay (MAD) was used as the quantitative measure of impulsivity. In Experiment 1, MADs were analyzed for sex differences. In Experiment 2, acquisition of cocaine self-administration was examined in rats selected for high (HiI; MADs < or =9 seconds) or low (LoI; MADs > or =13 seconds) impulsivity. In Experiment 3, HiI and LoI groups were compared on maintenance and extinction of cocaine self-administration and cocaine-primed reinstatement of drug-seeking behavior. There were no sex differences in impulsive choice; however, HiI male and female rats acquired cocaine self-administration faster than their LoI counterparts. LoI females responded more on a cocaine-associated lever during maintenance and extinction than HiI females, but HiI females showed greater reinstatement of cocaine-seeking behavior than all other groups at the highest dose tested (15 mg/kg). Thus, individual differences in impulsive choice were associated with differences in cocaine-seeking behavior. Impulsive choice and sex may be additive vulnerability factors in certain phases of drug abuse.