Heart failure (HF) is the inability of the heart to fill with or pump out enough blood to meet the body's needs. It is not one single disease, but rather a group of signs and symptoms caused by many different disease processes that have weakened the heart over time and left it unable to pump blood efficiently. Hypertension, diabetes mellitus, cardiovascular disorders are few of such disease processes responsible for conditions in HF. Even though, the co-morbidities mentioned above are well-established in the present system of medicine, its association with respiratory risk on obese patients especially in HF, still needs to be explored. The aim of this study is to determine the presence of dyspnea on exertion (DOE) in patients of HF as a consequence of obesity. Strategies to prevent the risk of HF, which would complement the current approaches aimed at Ayurvedic perspective especially, the obesity, its related comorbidities and contributors in the form of information on life-style leading to obesity needs to be focused. An ethical clearance for the project from the same institute was obtained on 101 patients of HF. All patients with ejection fraction <50% having DOE, New York Heart Association category were selected. Those with restrictive cardiomyopathy valvular abnormalities and under psychiatry treatment were excluded. Patients were informed about the project and their written consent was obtained followed by filling the Case Report Form (CRF). Their recent reports of left ventricular ejection fraction were attached along with details of 6 min hall walk test. Analysis was performed using the Statistical Package for the Social Sciences software, IBM version-17.0. The significant outcomes on lifestyles of HF related to co-morbidities were found.

Vata is one of the fundamental patho-physiological entities with unique and dynamic properties. All actions inside the body, whether voluntary or involuntary, are governed by Vayu . Actions involved during natural physiological calls or urges, are involuntary, but partially under voluntary control. Classical texts from Ayurveda state that such natural urges (NU) should be strictly attended without disturbing their natural flow. Some urges can be intentionally or inadvertently initiated ( Udiran ) or suppressed ( Dhaaran ), redirecting the normal flow of Vayu , leaving scopes for severe morbidities in the heart causing cardiovascular disorders (CVD). Since Vata has unique attribute of Vega (locomotion) that moves in a specific direction, its intensities can be quantified with the help of modern techniques. Few studies have objectively evaluated the intensities of NU like belching, sneezing, expulsion of flatus, etc. during normalcy, which may help us to determine their altered activity during morbidity. In spite of such studies, their relevance to CVD is inadequately explored. Hence, this article addresses details of such NU that lead to CVD alone, from classical texts of Ayurveda, conventional medicines and technology that quantify their intensities. Citing research articles from various journals using keywords were done to understand their mechanism along with their intensities. It was found that objective estimation of few NU was performed extensively whereas some had limitations. Theories from the classical texts confirm that physiological NU, if allowed to flow freely without any impedance, assures good health. It would certainly benefit the mankind if their pathologic state is timely detected so as to prevent disease progression in CVD.

A systems approach to health is the hallmark of Ayurveda. It believes in preventing disease and maintaining and restoring health. The entire concept stands on three fundamental functional units-Vata, Pitta and Kapha, where Vata, mobilizes the other two units. Depending on their locations, Vata (Vayu) is classified into five subtypes, where each has its distinct role to perform. Vyana Vayu (VV), an important subtype of Vata, is synthesized in myocytes and responsible for the genesis of the action potential. A key regulator in contractile functions, VV propels out nutrients from the heart. It not only mediates intracrine and paracrine activities but modulates the vascular tone too. Wherever there is scope to flow, VV has its unique role to contribute. Ancient scholars of Ayurveda have identified its ubiquitous role in the endogenous system, where all the activities depend on VV. Hence, preventing VV from any stimulus is of paramount importance since they consequently lead to various cardio vascular diseases (CVD). Classical texts have addressed the prognosis in six discrete phases where each phase can be avoided strategically. Highlighting the precipitants that attenuate VV, we focus on addressing those phases along with curative measures so that the functions of Vyana Vayu can be restored.