Diffuse duodeno-jejunal hemangiomatosis in children is a rare cause of bilious vomiting. In the clinical approach to bilious vomiting, tumors of the duodenum come at the end of the differential list--not to mention the rarity of hemangiomatosis. To our knowledge, isolated duodeno-jejunal hemangiomatosis as a cause of bilious vomiting in children is being reported for the first time. We analyse the various imaging modalities available to reach a clinical diagnosis.

Background:  Vascular complications are fairly common in patients suffering from Behcet's disease. Peripheral arterial aneurysms affect nearly every artery in association with this disease. This report discusses the management of this complication in such patients. Patients and Methods:  Two young patients were referred to the Vascular Surgery Service, because of arterial aneurysms. Both patients were known to suffer from Behcet's disease and were on colchicine. Both had history of deep vein thrombosis and were on long‐term anticoagulant therapy (warfarin). Case 1: A 38‐year‐old man presented with recurrent abdominal pains which prove to be due to a saccular abdominal aortic aneurysm (AAA) of 6 cm diameter. An elective open repair of the AAA with tube Dacron graft was performed. At 1 year follow up the patient remained free of symptoms. Case 2: A 42‐year‐old man presented with a neck swelling, which was confirmed by duplex scan and magnetic resonance angiography to be a saccular aneurysm of the left internal carotid artery. Surgical excision of the aneurysm was carried out and the artery repaired with a GORE‐TEX patch. Results:  At 12–18 months follow up, both patients remained well and asymptomatic. Conclusions:  Patients suffering from Behcet's disease are liable to get arterial aneurysms. Surveillance by non‐invasive studies ensures early diagnosis and management of these uncommon complications of the disease.

Objective and Importance: To describe the imaging findings of two cases of cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) mimicking multiple sclerosis. Clinical Presentation and Intervention: Two cases presenting with neurological signs and symptoms were referred for magnetic resonance imaging (MRI) evaluation of the brain. Case 1 was a 36-year-old female patient presenting with recurrent headaches and recent onset numbness in the fingers of the right hand. Neurological examination showed a mild sensory deficit in the right hand. Case 2 was a 31-year-old female patient presenting with attacks of right-sided numbness of the face and body. The neurological examination revealed a sensory loss in the face and brisk deep tendon reflexes. Routine MRI sequences showed two types of lesions in both cases: ‘punctate’ hyperintense lesions on T₂-weighted images (T₂WI)/fluid-attenuated inversion recovery (FLAIR) images, hypointense on T₁-weighted images (T₁WI) and ‘diffuse’ white matter lesions, hyperintense on T₂WI/FLAIR sequences and isointense to hypointense on T₁WI. All lesions showed no contrast enhancement. Both cases were previously clinically and radiologically diagnosed as multiple sclerosis. There was a strong family history consistent with recurrent infarctions in other family members of both patients. Both cases were later diagnosed as CADASIL by skin biopsy/genetic linkage studies and follow-up. Conclusion:The cases showed that CADASIL causes stroke-like episodes in adults and can mimic multiple sclerosis on imaging. Clinical evaluation and MRI findings allow a differentiation of the two entities.

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This article presents fibrosing mesenteric tuberculosis in a 19-year-old Arab boy who presented with weight loss, fever, abdominal pain, and distension. Abdominal contrast enhanced computed tomography (CECT) was performed which showed large infiltrative ill-defined mesenteric-based enhancing soft tissue phlegmonous mass with surrounding desmoplastic reaction causing retraction-kinking of small bowel loops associated with central necrotic mesenteric lymph nodes, multifocal small bowel wall thickening, and ascites. Abdominal tuberculosis is a diagnostic challenge particularly if pulmonary tuberculosis is absent as in this case. CT appears to be the modality of choice if clinical and epidemiological suspicion is high in order to ensure early treatment for a favorable outcome.