Sleep bruxism (SB) is defined as a centrally mediated, non-peripheral, involuntary condition, characterised by increased mandibular activity, regardless of dental contacts. These definitions have been added to the classic concept of bruxism, such as the habit of grinding or clenching teeth. 1 According to its multifactorial etiology, SB is classified as primary, with no apparent cause, or secondary, related to other comorbidities. 2,3 As secondary etiological factors, associations with