We prospectively evaluated the results of plantar pressure measurement in 32 patients (43 feet) who had a proximal crescentic osteotomy of the first metatarsal with a modified McBride procedure. The procedure's effectiveness in increasing weightbearing under the first ray, decreasing pressure under the second metatarsal head, and the relationship of radiographic measurements of first metatarsal length and position to postoperative pressure measurements were evaluated. Mean followup was 29 months. Average peak pressure increased postoperatively under the second metatarsal head. Almost identical numbers of feet had first metatarsal elevation (12) or depression (11) greater than 2 mm. Radiographic evidence of first metatarsal elevation, but not shortening, was associated with diminishing peak pressure and pressure-time integral under the first metatarsal head and hallux. Five feet that had first metatarsal elevation greater than 2 mm had new second metatarsal transfer lesions develop. Eleven feet preoperatively and nine feet postoperatively had symptomatic second metatarsal pressure lesions. One lesion persisted, 10 resolved, and eight new lesions developed. Control of the crescentic osteotomy in the sagittal plane was unpredictable despite modification of the surgical technique to plantarly displace the distal segment of the first meta-tarsal. Although average second metatarsal pressure increased postoperatively, there was variability in the correlation of radiographic change and pedobarographic measurements.
Cryoballoon ablation in conjunction with the novel AC is feasible, safe, and most importantly affords PVI in nearly all veins without having to switch to a regular guidewire. However, RT recordings could be documented in only 47% of pulmonary veins.
Background: Left bundle branch area pacing (LBBAP) has recently been introduced as a novel physiological pacing strategy. Within LBBAP, distinction is made between left bundle branch pacing (LBBP) and left ventricular septal pacing (LVSP, no left bundle capture). Objective: To investigate acute electrophysiological effects of LBBP and LVSP as compared to intrinsic ventricular conduction. Methods: Fifty patients with normal cardiac function and pacemaker indication for bradycardia underwent LBBAP. Electrocardiography (ECG) characteristics were evaluated during pacing at various depths within the septum: starting at the right ventricular (RV) side of the septum: the last position with QS morphology, the first position with r’ morphology, LVSP and—in patients where left bundle branch (LBB) capture was achieved—LBBP. From the ECG’s QRS duration and QRS morphology in lead V1, the stimulus- left ventricular activation time left ventricular activation time (LVAT) interval were measured. After conversion of the ECG into vectorcardiogram (VCG) (Kors conversion matrix), QRS area and QRS vector in transverse plane (Azimuth) were determined. Results: QRS area significantly decreased from 82 ± 29 µVs during RV septal pacing (RVSP) to 46 ± 12 µVs during LVSP. In the subgroup where LBB capture was achieved (n = 31), QRS area significantly decreased from 46 ± 17 µVs during LVSP to 38 ± 15 µVs during LBBP, while LVAT was not significantly different between LVSP and LBBP. In patients with normal ventricular activation and narrow QRS, QRS area during LBBP was not significantly different from that during intrinsic activation (37 ± 16 vs. 35 ± 19 µVs, respectively). The Azimuth significantly changed from RVSP (−46 ± 33°) to LVSP (19 ± 16°) and LBBP (−22 ± 14°). The Azimuth during both LVSP and LBBP were not significantly different from normal ventricular activation. QRS area and LVAT correlated moderately (Spearman’s R = 0.58). Conclusions: ECG and VCG indices demonstrate that both LVSP and LBBP improve ventricular dyssynchrony considerably as compared to RVSP, to values close to normal ventricular activation. LBBP seems to result in a small, but significant, improvement in ventricular synchrony as compared to LVSP.
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