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E3V4M6 on the Glasgow Coma Scale) and slight neck rigidity were present. Neurological and general findings showed no other abnormality. Laboratory data on admission were erythrocyte sedimentation rate 109 mm/1h; white blood cells (WBC) 11,170/mm 3 ; and C-reactive protein (CRP) 6.3 mg/dl. Other laboratory tests, including HbA1c and urine analysis, were normal. HIV antibody was negative. Computerized brain tomography (CT) showed slight brain edema. The open pressure was raised to 300 mmH 2 O on lumbar puncture on admission. Her cerebrospinal fluid (CSF) was cloudy with 1833/mm 3 WBC (78.5 % polymorphonuclear cells), 44 mg/dl glucose (simultaneous blood sugar 130 mg/dl), and 344 mg/dl protein.A chest radiograph and cardioechograph were normal. Her CT of the abdominal, chest, and cervical regions revealed no focus of infection or malignancy. No CSF leakage or sinusitis was found in otolaryngological medical examinations. We immediately started the antibiotics ceftriaxone and vancomycin with dexamethazone, the standard therapy for acute bacterial meningitis. Gram staining of her CSF showed gram negative rods. Because on hospital day two a culture grew the pseudomonas genus, she was placed on ceftazidime and gentamycin. Later, the P. putida found in both her CSF and blood cultures was susceptible to ceftazidime, gentamycin, meropenem and imipenem. Her consciousness slowly improved, becoming clear on hospital day four. Her laboratory data for hospital day two showed a WBC count of 12090/ mm 3 and a CRP of 17.6 mg/dl. In the CSF study performed on the same day, her WBC count was 7753/mm 3 . Subsequent blood and CSF tests showed improvement, and no bacteria were detected in her CSF. Because drug-induced