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Acquired septa1 defect secondary t o myocardial infarction can no longer be considered a rarity. 100 cases are reported in a recent paper of Sahagun and Burns, 132 by Sanders e t al., and a thorough scanning of the literature including less wellknown worli from eastern Europe revealed 79 described since the earlier review of Bond e t al. containing 93 references, the total number rising hereby t o 172 with a n ever increasing proportion of ante mortem diagnoses. Thus, mere addition of two more cases, even if diagnosed during life, would hardly be justified, unless they contribute t o the elucidation of certain features, anatomical and functional, of this condition. Case Reports.

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