B.1.1.7 is a recently discovered variant of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) associated with increased transmissibility. Recent findings indicate that this variant has a propensity to infect adolescents and children at higher rates than adults. The virus gains entry into various body cells utilizing angiotensin-converting enzyme 2 (ACE-2) and basigin (CD147) as receptors. The virus mainly affects type II pneumocytes of lungs, endothelial cells, enterocytes, and renal tubular cells. It is reported to affect testes, causing testicular pain, and producing histopathological changes, as observed in some autopsies. The B.1.1.7 variant can also affect various cells in the testes. This raises a major concern regarding the long-term effects of the viral infection on spermatogenesis and highlights the pressing need for a robust database of serum samples from infected male children.
BACKGROUND: Multiple variations of the palmaris longus (PL) muscle are reported throughout literature and include variations such as double muscle bellies, inversion of muscle belly and tendon, and the absence of the muscle. The muscle functions as a weak flexor of the wrist. It originates from the medial epicondyle of the humerus, projects as a superficial muscle of the anterior compartment of the forearm, lies over the transverse carpal ligament, and inserts into the palmar aponeurosis.
CASE REPORT: Routine dissection of the right forearm of a 70-year-old Caucasian male revealed a rare duplicated PL muscle with the medial PL muscle being reversed. The left forearm appeared normal, with no such variations. The cadaver showed no significant pathological findings due to this variation. Innervation and vasculature of the variant muscle appeared normal.
CONCLUSION: The PL muscle is highly variable, and clinicians need to be aware of its many possible presentations due to its involvement in surgery, symptomatic clinical presentations, and as an anatomical landmark.
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