Purpose:To study the clinical profile of patients undergoing scleral buckle removal (SBR) surgery.Materials and Methods:All consecutive patients undergoing SBR surgery following scleral buckle for retinal detachment repair between January 2002 and December 2011 with a minimum postSBR follow-up of 6 months were included in this study. A record based on analysis of indications, methods, complications, and outcomes of the eyes was performed.Results:One hundred and two eyes of 101 patients (men = 77; 76.24% and women = 24; 23.76%) belonging to the age group of 15–78 years (mean 50 ± 15 years) were included in this study. Time gap between scleral buckle and SBR ranged from 2 to 216 months (mean 61 ± 51 months). Buckle exposure with clinical infection (81; 79.41%) was the most common indication. Of 90 (88.2%) eyes with positive culture, 75 (83.3%) revealed single and 15 (16.6%) revealed multiple microorganisms. Staphylococcus epidermidis (42; 41.2%), was the most common isolate. Fungus was isolated in 3 (2.94%) eyes. Globe perforation (14; 13.7%) and recurrent retinal detachment (7; 6.9%) were the commonest complications. Time gap between SBR and recurrent retinal detachment ranged from 15 days to 50 months (mean 12.2 ± 18.3 months).Conclusions:Most of the exposed scleral buckles developed clinical infection few months to years after surgery, ultimately requiring SBR. Recurrent retinal detachment after SBR may appear from few days to years later warranting a long-term follow-up.

A lens coloboma is characterized by the lens tissue's notching at the equator. It is usually inferior-nasal else it is called an atypical coloboma. We report a young male who presented with a superior lens coloboma, an elevation deficit and moderate ptosis, left eye. As superior rectus and levator muscles are derived from the neural crest cell we propose, genetically, there may be an association between the lens coloboma and the superior rectus/levator muscle underaction via an anomalous homeobox gene function. RESUMENUn coloboma de cristalino se caracteriza por la presencia de un orificio en el ecuador del tejido del cristalino. Suele ser de tipo inferonasal; en caso contrario se denomina coloboma atípico. Informamos aquí de un varón joven que presentaba en el ojo izquierdo un coloboma de cristalino superior, un déficit de elevación y ptosis moderada. Puesto que el músculo recto y el elevador del párpado se desarrollan a partir de células de la cresta neural, proponemos la hipótesis de que pudiera haber una relación, desde el punto de vista genético, entre el coloboma de cristalino y el mal funcionamiento del músculo recto superior y del elevador, derivado todo de un funcionamiento anómalo de alguno de los genes de homeosecuencia. (J Optom 2009;2:67-69 ©2009 Consejo General de Colegios de Ópticos-Optometristas de España) PALABRAS CLAVE: coloboma del cristalino; ptosis; parálisis.