that only two patients in a group of 34 stroke patients with aphasia had totally recovered language six months post-onset. In later stages, the percentage of patients who were fully recovered is higher. It was shown that about one third of stroke survivors with aphasia have recovered from aphasia 12 to 18 months post-onset, whereas about 60% remain chronically aphasic [2,5,6]. According to symptoms and localization of the brain lesion, aphasia can be categorized into different syndromes. The classical syndromes of aphasia as defined by the Boston school [7] are: Global aphasia, Broca's aphasia, Wernicke's aphasia, conduction aphasia, anomic aphasia, transcortical motor aphasia, transcortical sensory aphasia and mixed transcortical aphasia [3,8,9], though this classification has given way to more recent emphasis on patterns of breakdown within cognitive, neuropsychological models of language breakdown and more fine-grained lesion studies [10,11]. However, regarding the objective of this paper, the focus is on individuals with Broca's aphasia and conduction aphasia as defined by Goodglass, et al. based on the Boston Diagnostic Aphasia Examination [7]. Broca's Aphasia is said to be characterized by non-fluent, sparse and effortful speech output, with reduced phrase length and syntactic complexity, and awkward articulation [3,9]. The dominant feature of Broca's aphasia is agrammatism. During language production, agrammatic patients predominantly use content words (nouns and main verbs), giving the well-known impression of telegraphic speech, and associated problems producing full sentences. In its more severe form, spoken utterances may be reduced to single words. Patients with Broca's aphasia also have word-finding difficulties,
