Aims and Objectives The purpose of this study was to understand the emotional intelligence level (EI) and negative emotional status of the front‐line nurses in the epidemic situation and to further explore the relationship between them. Background During the COVID‐19 epidemic, under the influence of multiple factors, nurses were vulnerable to negative emotions. While previous studies have explored, the role of emotional intelligence in negative emotions, the relationship between the two has not been sufficiently discussed in the context of COVID‐19. Design The study carried out a cross‐sectional survey. The STROBE was selected as the checklist in this study. Methods 202 nurses from Wuhan makeshift hospital participated in the questionnaire survey. Data collection tools included a general data questionnaire designed by the researchers, Chinese version of EI scale (WLEIS‐C) and Chinese version of Depression Anxiety Stress Scale (DASS‐21). Descriptive statistics, single factor analysis and correlation analysis were used to analyse the data. Results The emotional intelligence of the front‐line nurses was in the upper middle range. Among the negative emotions, anxiety was the most prominent symptom. Conclusions Managers should pay attention to the negative emotional problems of front‐line nurses, improve their EI level and promote mental health and the progress of epidemic prevention. Relevance to clinical practice Improving the level of emotional intelligence can reduce the frequency and intensity of negative emotions. In clinical work, emotional intelligence can be used as a skill to carry out relevant training, which is conducive to playing a positive role in future emergencies.

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This research found an absence of a systematic guideline development methodology and a tendency to rely on expert opinion in the process of developing NPGs. The readers should consider these flaws and limitations when using recommendations in those NPGs.

Abstract:1 The hygroscopic growth of aerosol particles is a key factor of air pollution because it 2 can significantly reduce visibility. In order to better understand the impact of the 3 hygroscopic growth effect on haze events and contributing factors, we made use of rich 4 measurements during an intensive field campaign conducted in Xingtai, Hebei province 5 of China that has suffered from the most serious pollution in the Northern China Plain. 6Key measurements are from Raman lidar and ground-based instruments such as a 7GrayWolf 6-channel handheld particle/mass meter for atmospheric particulate matter 8 that have diameters less than 1 µm and 2.5 µm (PM1 and PM2.5, respectively), aerosol The aerosol acidity value of Case II (1.50) was greater than that of Case I (1.35) due to

Periplocin, as one of the components of cardiac glycosides in Cortex periplocae, exhibited cardiotonic effects. Orally ingesting periplocin in high doses or over prolonged periods would cause serious adverse reactions, especially cardiotoxicity, which limits the applications of periplocin in clinical therapy. It has been reported that Panax notoginseng saponins could be used in compatibility with periplocin to reduce the cardiotoxicity of periplocin. To clarify the mechanisms of periplocin-induced cardiotoxicity and compatibility-pairing in reducing cardiotoxicity, the gas chromatography-mass spectrometry method was used to detect and analyze the metabolic profiles of rat plasma and urine samples after oral administration of periplocin, Panax notoginseng saponins, and the different compatibility ratios of periplocin and Panax notoginseng saponins. The multivariate statistical analysis method was used to screen and identify the biomarkers. A total of 49 potential biomarkers (28 in plasma and 21 in urine) associated with periplocin-induced cardiotoxicity were identified. Seven pathways were found through metabolomic pathway analysis. Moreover, the levels of 42 biomarkers (22 in plasma and 20 in urine) were close to normal after compatibility pairing. By analyzing the relative metabolic pathways, Panax notoginseng saponins could effectively reduce the cardiotoxicity of periplocin by affecting the tricarboxylic acid cycle, energy metabolism, and arachidonic acid metabolism.