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Carbonaceous materials have been usually adopted as the anode for high energy density potassium-ion hybrid capacitors (KICs) owing to their low voltage plateau, high conductivity, and excellent electrochemical compatibility. The improvements of their specific capacity and sluggish intercalation mechanism are still challenges to further boost the energy density of KICs while maintaining high power density and long cycle life. Herein, a N-doped mesoporous carbon sphere array composite is developed by a dual-templates method. The N-doped carbon sphere array with interpenetrated macro-and meso-pores facilitates the fast electron transport and rapid K + diffusion. The uniformly introduced Co 3 O 4 nanoparticles (NPs) confined in the array enable a kinetically boosted conversion reaction for excess and fast K + storage. The partially oxidized Co NPs can efficiently enhance the conductivity of the entire composite. By introducing this optimized conversion capacity from encapsulated Co 3 O 4 NPs, the composite with intercalation and conversion coupling mechanisms displays superior capacity and cycle life. The assembled KICs exhibit high energy/power densities (148 Wh kg −1 /124 W kg −1 ) and great cycling performance (94% after 5000 cycles, 0.5 A g −1 ). This promising strategy demonstrates an example for carbonaceous composite anode with synergistic K + storage hybrid mechanism toward high performance KICs.

Congenital cataract disease is a clinically and genetically heterogeneous lens disorder. The purpose of this study was to identify the genetic defects and to investigate the relationships between disease-causing genes and lens morphology in congenital cataracts. Patients were given a physical examination, and their blood samples were collected for DNA extraction. Mutation analysis was performed by direct sequencing of the following candidate genes: CRYGC, CRYGD, CRYGS, GJA8, GJA3 and CRYAA. Mutational analysis of CRYGD identified a recurrent (p.P24T) mutation in two unrelated families with congenital coralliform cataracts and three novel (p.Q101X, p.E104fsX4 and p.E135X) mutations in three families with congenital nuclear cataracts. The p.E135X mutation is a de novo mutation. Haplotype analysis showed patients inherited the same CRYGD allele originated from father. The p.E135X mutation seen in two siblings suggests a mechanism of gonadal mosaicism in the father.

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