Numerous investigators have tested contentions that angry faces capture early attention more completely than happy faces do in the context of other faces. However, syntheses of studies on early event‐related potentials related to the anger superiority hypothesis have yet to be conducted, particularly in relation to the N200 posterior‐contralateral (N2pc) component which provides a reliable electrophysiological index related to orienting of attention suitable for testing this hypothesis. Fifteen samples (N = 534) from 13 studies featuring the assessment of N2pc amplitudes during exposure to angry‐neutral and/or happy‐neutral facial expression arrays were included for meta‐analysis. Moderating effects of study design features and sample characteristics on effect size variability were also assessed. N2pc amplitudes elicited by affectively valenced expressions (angry and happy) were significantly more pronounced than those elicited by neutral expressions. However, the mean effect size difference between angry and happy expressions was ns. N2pc effect sizes were moderated by sample age, number of trials, and nature of facial images used (schematic vs. real) with larger effect sizes observed when samples were comparatively younger, more task trials were presented and schematic face arrays were used. N2pc results did not support anger superiority hypothesis. Instead, attentional resources allocated to angry versus happy facial expressions were similar in early stages of processing. As such, possible adaptive advantages of biases in orienting toward both anger and happy expressions warrant consideration in revisions of related theory.

Theories of pain-related attention biases (ABs) have posited that people are predisposed to focus upon pain because prioritizing attention towards potential threats enhances survival (Eccleston & Crombez, 1999). Unfortunately, when ongoing pain is an attentional priority over extended intervals, healthy behaviours may be compromised, impairment may be compounded and pain chronicity may become entrenched (Vlaeyen & Linton, 2000). In sum, pain-related ABs are hypothesized to increase risk for the development and maintenance of chronic pain. Meta-analyses based on reaction times (RTs) to stimuli from laboratory tasks have found selective support for these premises (Crombez, Van Ryckeghem, Eccleston, & Van Damme, 2013; Todd, van Ryckeghem, Sharpe, & Crombez, 2018). For example Todd et al. (2018) found chronic pain samples showed modestly