Apatite fi ssion-track thermochronology data elucidate the cooling/exhumation history of the Qinling (Qin Mountains), which contain a Paleozoic−Mesozoic orogenic collage north of the Sichuan Basin and northeast of the Tibetan Plateau. In particular, we examine the extent to which the Qinling were affected by the rising plateau. The northern and eastern Qinling show continuous cooling and slow exhumation since the Cretaceous. In contrast, in the southwestern Qinling, rapid cooling initiated at 9−4 Ma, a few million years later than in the eastern Tibetan Plateau. A compilation of major Cenozoic faults in the eastern Tibetan Plateau and the Qinling, and their kinematic and dynamic characterization, shows that deformation in the Qinling has predominantly been strike slip. Active sinistral and dextral strike-slip faults delineate an area of eastward rock fl ow and bound the area of rapid late Cenozoic cooling outlined by apatite fi ssion-track thermochronology. These data can be interpreted to indicate that lower crustal fl ow has been diverted around the Longmen Shan and beneath the southwestern Qinling, causing active plateau uplift in this area. Alternatively, northeastern Tibet may be growing eastward faster in the western Qinling than the entire South China Block is extruding to the east.

Our results strongly suggest that nano-TiO(2) has an obvious impact on biomolecules. Our data suggest that more attention should be paid to the potential toxicity of nano-TiO(2) on biomolecules. Further research into the toxicity of nanosized particles needs to be carried out prior to their cell toxicity and tissue toxicity. These investigations might serve as the basis for determining the toxicity and application of nanomaterials.

Background: Health risk from exposure of perfluorochemicals (PFCs) to wildlife and human has been a subject of great interest for understanding their molecular mechanism of toxicity. Although much work has been done, the toxigenicity of PFCs remains largely unknown. In this work, the noncovalent interactions between perfluorooctane sulfonate (PFOS) and serum albumin (SA) and DNA were investigated under normal physiological conditions, aiming to elucidate the toxigenicity of PFCs.

No abstract