Background & objectives: Conventional methods are not an efficient method in the removal of resistant organic pollution. Ozone molecules in the presence of anion persulfate can be used as an appropriate method for the removal of these pollutants. The aim of this study is to determine the efficiency of the combined process of ozone and radical sulfate in decomposition of SDBS and reduce the concentration of this pollutant in aqueous. Methods: In this experimental study, use of semi-batch reactor by one liter volume was used semi-continuously. The effect of pH (3-11), concentration of persulfate (10-100 mM/L), concentration of O 3 (1-5 mg/L.hr) and initial concentration of SDBS (10-100 mg/L) were investigated. The kinetics of the reaction, effect of radical scavenger and COD removal in the proper conditions of the process was determined. The concentration of SDBS and COD were measured using a standard reference method. Results: The efficiency of process in 40 minute was more than 97 percent while the process parameters were pH=3, initial concentration of SDBS was 10 mg/L, concentration of O 3 was 5 mg/L.hr and persulfate anions was 20 mM/L. By changing the parameters and the presence of radical scavenger, process efficiency decreased. The efficiency of COD removal in 70 minutes was 80 percent. The reaction kinetic followed by first order kinetic. Conclusion: The ozonation process in the presence of persulfate anion due to the production of active persulfate radical can be suitable method for the removal of POPs such as SDBS. By this method, it is possible to increase the treatment of the wastewater containing this pollutant and reduce the organic loading to environment.

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Background: Dyskeratosis congenita, an inherited and rare disease prevalent in males, is clinically manifested by reticulate hyperpigmentation, nail dystrophy, and leukoplakia. DC is associated with the increased risk of malignancy and other potentially lethal complications such as bone marrow failure, as well as lung and liver diseases. Mutations in 19 genes were found to be correlated with DC. Herein, we report a 12-year-old boy carrying a de novo mutation in TINF2 gene. Methods: WES was performed on DNA sample of the proband, and the variant was investigated in the family by Sanger sequencing. Population and bioinformatics analysis were performed. Results: The NM_ 001099274.3( TINF2 ): c.844C>T (p.Arg282Cys) mutation was found by WES. Conclusion: There was no history of the disease in the family, and the variant was classified as a de novo mutation.