538. Differentiation of alcoholics. Childhood history of minimal brain dvsfunction. familv historv. and drink-ing pattern

Tarter, Ralph E.; Mcbride,; Herbert,; Buonpan, E; Schneider, Jean-Guy

doi:10.1097/00006199-197711000-00073

Cited by 33 publications

(39 citation statements)

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“…Behavioral ratings of attentiondeficit hyperactivity symptoms were obtained using items selected from previous behavioral checklists (Tarter, McBride, Buonpane, Dorothea, & Schneider, 1977;Wender, 1971) querying behavior prior to age 12. More information on these scales is available in Hesselbrock (1986).…”

Section: Methodsmentioning

confidence: 99%

Executive–cognitive functioning in the development of antisocial personality disorder

Stevens

Kaplan

Hesselbrock

2003

Addictive Behaviors

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Section: Methodsmentioning

confidence: 99%

Executive–cognitive functioning in the development of antisocial personality disorder

Stevens

Kaplan

Hesselbrock

2003

Addictive Behaviors

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“…Early studies found greater prevalence of alcoholism in biological mothers and fathers of adopted hyperactive children, but not the legal parents (Cantwell, 1975), indicating a substantial biological component mediating this relationship. Retrospective reports of hyperactivity in alcoholic men predicted earlier onset of alcoholism and more family history of alcohol problems (Tarter, McBride, Buonpane, & Schneider, 1977). More recently, Kuperman, Scholssner, Lidrad, and Reich (1999) found that the children with alcoholic parents had a higher likelihood of having ADHD after controlling for family factors such as low income and child-parent conflict.…”

Section: Children Of Alcoholicsmentioning

confidence: 98%

Inattention/hyperactivity and aggression from early childhood to adolescence: Heterogeneity of trajectories and differential influence of family environment characteristics

Jester

Nigg

Adams

et al. 2005

Develop. Psychopathol.

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In attention/hyperactivity and aggressive behavior problems were measured in 335 children from school entry throughout adolescence, at 3-year intervals. Children were participants in a high-risk prospective study of substance use disorders and comorbid problems. A parallel process latent growth model found aggressive behavior decreasing throughout childhood and adolescence, whereas inattentive/hyperactive behavior levels were constant. Growth mixture modeling, in which developmental trajectories are statistically classified, found two classes for inattention/hyperactivity and two for aggressive behavior, resulting in a total of four trajectory classes. Different influences of the family environment predicted development of the two types of behavior problems when the other behavior problem was held constant. Lower emotional support and lower intellectual stimulation by the parents in early childhood predicted membership in the high problem class of inattention/hyperactivity when the trajectory of aggression was held constant. Conversely, conflict and lack of cohesiveness in the family environment predicted membership in a worse developmental trajectory of aggressive behavior when the inattention/hyperactivity trajectories were held constant. The implications of these findings for the development of inattention/hyperactivity and for the development of risk for the emergence of substance use disorders are discussed.A cluster of child problems including inattention, disorganization, hyperactivity, and impulsivity present serious dilemmas for our society. These problems tend to co-occur and, when sufficiently severe, are diagnosed clinically as attention-deficit/hyperactivity disorder (ADHD; American Psychiatric Association [APA], 2000). Clinical diagnosis of ADHD is associated with serious impairment in multiple domains (Johnston, Murray, Hinshaw, Pelham, & Hoza, 2002) and risk for poor long-term outcome (Klein & Mannuzza, 1991) including development of later substance use disorders (Zucker & Gomberg, 1986). Treatment of attention problems and hyperactivity has led to increased rates of medication of children in the United States (Robison, Sclar, Skaer, & Galin, 1999) and associated controversy (Searight & McLaren, 1998). As a result, understanding the antecedents of these problems is an important priority.Yet the determinants of this cluster of childhood problems are far from well understood. Twin and adoption studies indicate that heritability of normal and severe levels of these behavior problems is substantial (Loney, Paternite, Schwartz, & Roberts, 1997) and neuropsychological correlates are becoming more clearly delineated (Nigg, 2001). At the same time, it is recognized that epigenetic processes, including family context effects, must also be better understood to NIH-PA Author ManuscriptNIH-PA Author Manuscript NIH-PA Author Manuscript fully account for the developmental trajectory of these difficulties (Whalen & Henker, 1999). Indeed, even heritable processes are likely to exert at least part of the...

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“…Recently, it has been suggested that this syndrome is only apparently time limited and restricted to childhood, as previously thought [Läufer and Dennhoff, 1967], but may persist in adults with variable clinical features. Sev eral prospective, retrospective, and family studies have found in ADD a higher prevalence of various adult psy chiatric disorders, principally sociopathy [Borland, 1978;Morrison, 1979;Milman, 1979], psychoses [Menkes et al, 1967;Beliak, 1976;Mednick and Schulsinger, 1968], and alcoholism [Tarter et al, 1977]. Furthermore, other au thors found an association between some characters of adult psychopathology (i.e., anxiety, impulsivity, distractibility, aggressive behaviour) and a probable anamnestic diagnosis of childhood hyperkinesis [Hartocollis, 1968;Mendelson et al, 1971;Mann and Greenspan, 1976;Mor rison, 1979;Wender et al, 1981].…”

mentioning

confidence: 99%

EEG and Clinical Findings during Pemoline Treatment in Children and Adults with Attention Deficit Disorder

Caresia¹,

Pugnetti²,

Besana

et al. 1984

Neuropsychobiology

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A clinical study has been conducted on a selected group of 9 children and 12 adults with attention deficit disorder, evaluating the response to the therapy with magnesium pemoline by means of EEG power spectra. Pemoline was given orally in increasing doses ranging from 20 to 80 mg/day. EEG was recorded during baseline and at three fixed intervals during the treatment. Clinical response was assessed by objective and subjective ratings. In the adult sample, the drug produced two opposite patterns of EEG response which allowed us to discriminate by the 1st day of treatment responders who reported a paradoxical calming and non-responders who reported a worsening in anxiety. Inconsistent EEG power changes produced by the drug in children did not allow the discrimination of responders and non-responders. Children did not show paradoxical signs of sedation. EEG and clinical data suggest that adults and children show different drug response and that the paradoxical sedation is present only in adults.

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538. Differentiation of alcoholics. Childhood history of minimal brain dvsfunction. familv historv. and drink-ing pattern

Cited by 33 publications

References 0 publications

Executive–cognitive functioning in the development of antisocial personality disorder

Executive–cognitive functioning in the development of antisocial personality disorder

Inattention/hyperactivity and aggression from early childhood to adolescence: Heterogeneity of trajectories and differential influence of family environment characteristics

EEG and Clinical Findings during Pemoline Treatment in Children and Adults with Attention Deficit Disorder

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