2020
DOI: 10.12659/ajcr.926737
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A 65-Year-Old Woman with a History of Type 2 Diabetes Mellitus and Hypertension and a 15-Day History of Dry Cough and Fever Who Presented with Acute Renal Failure Due to Infection with SARS-Cov-2

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Cited by 3 publications
(7 citation statements)
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“…While the infection was associated with an increased risk of acute kidney injury (125.4 events per 100,000 persons), vaccination with BNT162b2 mRNA vaccine could reduce the risk (−4.6 events per 100,000 persons) ( 103 ). The pathophysiology of SARS-CoV-2-related renal injury is presumed to be multifactorial through direct (i.e., virus or vaccine components) or indirect (i.e., immune-mediated like cytokine storm or hyperactivity of T cells) effects ( 104 ). After vaccination, suspension to kidney injury encompasses manifestations like oliguria or anuria, edema or anasarca, hypertension, and dyspnea due to pleural effusion ( 98 ).…”
Section: Renal Complicationsmentioning
confidence: 99%
“…While the infection was associated with an increased risk of acute kidney injury (125.4 events per 100,000 persons), vaccination with BNT162b2 mRNA vaccine could reduce the risk (−4.6 events per 100,000 persons) ( 103 ). The pathophysiology of SARS-CoV-2-related renal injury is presumed to be multifactorial through direct (i.e., virus or vaccine components) or indirect (i.e., immune-mediated like cytokine storm or hyperactivity of T cells) effects ( 104 ). After vaccination, suspension to kidney injury encompasses manifestations like oliguria or anuria, edema or anasarca, hypertension, and dyspnea due to pleural effusion ( 98 ).…”
Section: Renal Complicationsmentioning
confidence: 99%
“…Several case reports of acute kidney injuries in patients with or without prior renal pathology following vaccination [62][63][64] . The pathophysiology of SARS-CoV-2 related renal injury is presumed to be multifactorial through direct (i.e., virus or vaccine components) or indirect (i.e., immunemediated like cytokine storm or hyperactivity of T cells) effects 65 . After vaccination conducted by clinical symptoms, suspension to kidney injury encompass oliguria or anuria, edema or anasarca, hypertension, and dyspnea due to pleural effusion 60 .…”
Section: Renal Complicationsmentioning
confidence: 99%
“…Acute tubular necrosis can be presented with increased creatinine and urea nitrogen, proteinuria, hypoalbuminemia, biopsy findings of diffuse PCT injury, lymphocyte infiltration, and cell necrosis 60,61 . Furthermore, hypodensity of renal parenchyma may have been seen in computed tomography (CT) 65 . Treatment consists of two important approaches; firstly, kidney protection from further injury by adjusting input and output fluids, excluding nephrotoxic drugs, and monitoring creatinine level.…”
Section: Renal Complicationsmentioning
confidence: 99%
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“…If the symptomatic treatment is not timely, it will cause anxiety and depression symptoms of the patient, and cause damage to the eyes, nerves, heart, and other parts of the patient. What is more, serious comorbidities such as kidney failure, myocardium, and cerebral infarction will occur ( 11 , 12 ). At the same time, the symptoms of anxiety and depression will in turn affect the patient’s blood sugar and blood pressure control, making the symptoms of anxiety and depression more serious, and then forming a vicious circle.…”
Section: Introductionmentioning
confidence: 99%